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Merck
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Key Documents

GHS280

Sigma-Aldrich

Hematoxylin Solution, Gill No. 2

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About This Item

MDL number:
UNSPSC Code:
41116124
NACRES:
NA.47

form

solution

Quality Level

shelf life

Expiry date on the label.

IVD

for in vitro diagnostic use

concentration

4 g/L

pH

2.5-3.5

application(s)

hematology
histology

storage temp.

room temp

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Application

Gill formulation No. 2 may be used for cytology and/or histology as a progressive or regressive stain depending on length of staining time.

Other Notes

4 g/L certified hematoxylin

pictograms

Corrosion

signalword

Danger

hcodes

Hazard Classifications

Eye Dam. 1

Storage Class

10 - Combustible liquids

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable


Certificados de análisis (COA)

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H B Baker et al.
Tissue engineering. Part A, 23(11-12), 572-584 (2017-02-07)
Wounds to the head, neck, and extremities have been estimated to account for ∼84% of reported combat injuries to military personnel. Volumetric muscle loss (VML), defined as skeletal muscle injuries in which tissue loss results in permanent functional impairment, is
J A Passipieri et al.
Tissue engineering. Part A, 23(11-12), 556-571 (2017-02-09)
Volumetric muscle loss (VML) injuries exceed the considerable intrinsic regenerative capacity of skeletal muscle, resulting in permanent functional and cosmetic deficits. VML and VML-like injuries occur in military and civilian populations, due to trauma and surgery as well as due
László Potor et al.
International journal of molecular sciences, 21(13) (2020-07-09)
Hemoglobin, heme and iron are implicated in the progression of atherosclerosis. Therefore, we investigated whether the hydrophobic fungal iron chelator siderophore, desferricoprogen (DFC) inhibits atherosclerosis. DFC reduced atherosclerotic plaque formation in ApoE-/- mice on an atherogenic diet. It lowered the
Andrew L Wishart et al.
Science advances, 6(43) (2020-10-23)
The extracellular matrix (ECM), a major component of the tumor microenvironment, promotes local invasion to drive metastasis. Here, we describe a method to study whole-tissue ECM effects from disease states associated with metastasis on tumor cell phenotypes and identify the

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