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Merck

343226

Sigma-Aldrich

Nickel sulfide

99.7% trace metals basis, −150 mesh

Sinónimos:

Nickel subsulfide

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About This Item

Fórmula empírica (notación de Hill):
Ni3S2
Número de CAS:
Peso molecular:
240.21
EC Number:
MDL number:
UNSPSC Code:
12161600
PubChem Substance ID:
NACRES:
NA.23

assay

99.7% trace metals basis

form

powder

reaction suitability

reagent type: catalyst
core: nickel

particle size

−150 mesh

density

5.8 g/mL at 25 °C (lit.)

application(s)

battery manufacturing

SMILES string

[Ni]=S=[Ni]=S=[Ni]

InChI

1S/3Ni.2S

InChI key

YGHCWPXPAHSSNA-UHFFFAOYSA-N

General description

Major phase is Ni3S2. Product may also include a small amount of sulfur-rich phase. Nickel Sulfide (Ni3S2) exists as a heazlewoodite mineral in nature. The structure of Ni3S2 is hombohedral. Raman spectral analysis of Ni3S2 has been reported.

Application

Nickel sulfide was implanted in mice, to induce the growth of malignant fibrous histiocytomas.

Packaging

Packaged in glass bottles

signalword

Danger

Hazard Classifications

Acute Tox. 3 Inhalation - Aquatic Acute 1 - Aquatic Chronic 1 - Carc. 1A Inhalation - Muta. 2 - Skin Sens. 1 - STOT RE 1 Inhalation

target_organs

Lungs

Storage Class

6.1D - Non-combustible acute toxic Cat.3 / toxic hazardous materials or hazardous materials causing chronic effects

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Faceshields, Gloves, type P3 (EN 143) respirator cartridges


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X Mao et al.
Cancer letters, 161(1), 57-62 (2000-11-18)
Differential display polymerase chain reaction (DD-PCR) was used to analyze the differentially expressed genes from nickel-transformed human embryonic lung (HEL) cells (MRC-9 and IMR-90) and their control counterparts (non-treated). Two genes, MS515 and IC82, were confirmed by Northern blot analysis.
M Okamoto
Differentiation; research in biological diversity, 61(5), 285-292 (1997-08-01)
It is well known that urodeles have the most powerful regenerative capacities among vertebrates, but there is little realisation that they are extremely resistant to spontaneous or chemically induced tumours. Regeneration and carcinogenesis have been considered to be two sides
Reactive oxygen-induced carcinogenesis causes hypermethylation of p16 (Ink4a) and activation of MAP kinase.
Govindarajan B, et al.
Molecular Medicine, 8(1) (2002)
Joseph R Landolph et al.
Environmental health perspectives, 110 Suppl 5, 845-850 (2002-11-12)
In the past, exposure of workers to mixtures of soluble and insoluble nickel compounds by inhalation during nickel refining correlated with increased incidences of lung and nasal cancers. Insoluble nickel subsulfide and nickel oxide (NiO) are carcinogenic in animals by
Aaron Barchowsky et al.
The Journal of biological chemistry, 277(27), 24225-24231 (2002-04-30)
Inhalation of particulate nickel subsulfide (Ni3S2) causes chronic active inflammation and fibrosis of the lungs. However, the mechanisms for these effects are not well understood. Therefore, cell culture experiments with BEAS-2B human airway epithelial cells were conducted to test the

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