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Merck
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690228

Sigma-Aldrich

N,N-Bis[3-(methylamino)propyl]methylamine

98%

Sinonimo/i:

N-Methyl-3,3′-bis(methylamino)dipropylamine, N-Methyl-N,N-bis[3-(methylamino)propyl]amine, Methylbis(3-methylaminopropyl)amine

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About This Item

Formula condensata:
CH3N(CH2CH2CH2NHCH3)2
Numero CAS:
123-70-6
Peso molecolare:
173.30
Beilstein:
6711228
Numero CE:
204-645-0
Numero MDL:
MFCD00014857
Codice UNSPSC:
12352100
ID PubChem:
329761321
NACRES:
NA.22

Saggio

≥97.5% (GC)
97.5-102.5% (with HClO4, T)
98%

Stato

liquid

Impurezze

≤1% water

Stringa SMILE

CNCCCN(C)CCCNC

InChI

1S/C9H23N3/c1-10-6-4-8-12(3)9-5-7-11-2/h10-11H,4-9H2,1-3H3
PQYGBJHVVYREGU-UHFFFAOYSA-N

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Pittogrammi

Corrosion
GHS05

Avvertenze

Danger

Indicazioni di pericolo

H314

Classi di pericolo

Skin Corr. 1B

Codice della classe di stoccaggio

8A - Combustible corrosive hazardous materials

Classe di pericolosità dell'acqua (WGK)

WGK 3

Punto d’infiammabilità (°F)

Not applicable

Punto d’infiammabilità (°C)

Not applicable

Dispositivi di protezione individuale

Faceshields, Gloves, Goggles, type ABEK (EN14387) respirator filter

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Certificati d'analisi (COA)

Lot/Batch Number
1414612V
1414612

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Sigma-Aldrich

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Sigma-Aldrich

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N,N,N′,N′-Tetramethyl-1,4-butanediamine

Tris(2-aminoethyl)amine 96%

Sigma-Aldrich

225630

Tris(2-aminoethyl)amine

Gold thioglucose, mimpa and lesions of the ventromedial hypothalamus.
D F Brown
Journal of neuropathology and experimental neurology, 43(4), 455-457 (1984-07-01)
S Levine et al.
Journal of immunological methods, 54(3), 355-360 (1982-11-12)
In the past, the lesions of experimental allergic encephalomyelitis (EAE) have been induced to localize around brain tissue damaged by anoxia or direct physical or chemical attack. The procedure for producing the requisite antecedent brain injury has been simplified by
D Nochlin et al.
Journal of neuropathology and experimental neurology, 41(2), 233-240 (1982-03-01)
The administration of an aliphatic triamine, 3,3'-methylimino-bis-(N-methylpropylamine), was used to produce non-necrotizing lesions in the hypothalamus and medulla oblongata of rats. The distribution of the lesions in the hypothalamus was similar to that produced by goldthioglucose, and consisted ultrastructurally of
J C Lyons et al.
Journal of neuropathology and experimental neurology, 41(1), 45-53 (1982-01-01)
The clinical and pathologic features of two brothers with the adult form of Gaucher's disease, both of whom developed cerebral glioblastoma multiforme, are presented. Neither patient had a long-standing neurologic disorder, and morphologic evidence of nervous system glucocerebroside storage was
S Levine et al.
Brain research, 242(2), 219-225 (1982-06-24)
An aliphatic triamine has been reported to cause lesions in rats in the vicinity of the area postrema and the median eminence of the hypothalamus, sites known to lack a blood-brain barrier. The present study revealed that some of the
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