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MAB1548

Sigma-Aldrich

Anti-Myosin Antibody, heavy chain β

culture supernatant, clone 5B9 (2C8), Chemicon®

Synonym(s):

Anti-CMD1S, Anti-MPD1, Anti-MYHCB, Anti-SPMD, Anti-SPMM

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About This Item

UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41

biological source

mouse

Quality Level

100

antibody form

culture supernatant

antibody product type

primary antibodies

clone

5B9 (2C8), monoclonal

species reactivity

human

manufacturer/tradename

Chemicon®

technique(s)

immunohistochemistry: suitable
western blot: suitable

isotype

IgG2a

NCBI accession no.

NM_000257.2

UniProt accession no.

P12883

shipped in

wet ice

target post-translational modification

unmodified

Gene Information

rat ... Myh7(29557)

Specificity

Recognizes the S1/S2 junction of human ventricular myosin heavy chain beta

Immunogen

Epitope: heavy chain beta
Human ventricular myosin

Application

Detect Myosin using this Anti-Myosin Antibody, heavy chain β validated for use in WB, IH.
Immunohistochemistry: Neat (undiluted)
Western blot: 1:10

Optimal dilutions must be determined by the end user.

Physical form

Supernatant in RPMI 1640, 10% FCS, 0.01% sodium azide.

Storage and Stability

Maintain at -20°C in undiluted aliquots for up to 12 months. Avoid repeated freeze/thaw cycles.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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Storage Class Code

12 - Non Combustible Liquids

WGK

WGK 2

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Barbara S Mallon et al.
Stem cell research, 12(2), 376-386 (2014-01-01)
Many studies have compared the genetic and epigenetic profiles of human induced pluripotent stem cells (hiPSCs) to human embryonic stem cells (hESCs) and yet the picture remains unclear. To address this, we derived a population of neural precursor cells (NPCs)
Siva K Panguluri et al.
American journal of physiology. Heart and circulatory physiology, 304(12), H1651-H1661 (2013-04-16)
Ventricular arrhythmias account for high mortality in cardiopulmonary patients in intensive care units. Cardiovascular alterations and molecular-level changes in response to the commonly used oxygen treatment remains unknown. In the present study we investigated cardiac hypertrophy and cardiac complications in
Henry E Young et al.
The anatomical record. Part A, Discoveries in molecular, cellular, and evolutionary biology, 277(1), 178-203 (2004-02-26)
Undifferentiated cells have been identified in the prenatal blastocyst, inner cell mass, and gonadal ridges of rodents and primates, including humans. After isolation these cells express molecular and immunological markers for embryonic cells, capabilities for extended self-renewal, and telomerase activity.
Henry E Young et al.
The anatomical record. Part A, Discoveries in molecular, cellular, and evolutionary biology, 276(1), 75-102 (2003-12-31)
Development of a multicellular organism is accomplished through a series of events that are preprogrammed in the genome. These events encompass cellular proliferation, lineage commitment, lineage progression, lineage expression, cellular inhibition, and regulated apoptosis. The sequential progression of cells through
Richard P Davis et al.
Circulation, 125(25), 3079-3091 (2012-06-01)
Pluripotent stem cells (PSCs) offer a new paradigm for modeling genetic cardiac diseases, but it is unclear whether mouse and human PSCs can truly model both gain- and loss-of-function genetic disorders affecting the Na(+) current (I(Na)) because of the immaturity
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