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203149

Sigma-Aldrich

Copper(II) chloride

99.999% trace metals basis

Synonym(s):

Cupric chloride

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About This Item

Linear Formula:
CuCl2
CAS Number:
Molecular Weight:
134.45
EC Number:
MDL number:
UNSPSC Code:
12352302
eCl@ss:
38150111
PubChem Substance ID:
NACRES:
NA.23

Assay

99.999% trace metals basis

form

powder

reaction suitability

reagent type: catalyst
core: copper

impurities

≤15.0 ppm Trace Metal Analysis

mp

620 °C (lit.)

density

3.386 g/mL at 25 °C (lit.)

application(s)

battery manufacturing

SMILES string

Cl[Cu]Cl

InChI

1S/2ClH.Cu/h2*1H;/q;;+2/p-2

InChI key

ORTQZVOHEJQUHG-UHFFFAOYSA-L

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General description

Copper chloride is a white solid. It is insoluble in water. Copper(II) chloride may be prepared by dissolving excess of copper (II) oxide or copper(II) carbonate in dilute hydrochloric acid.

Application

Adding CuCl2 to AgNO3 with EG1mYAbe is used to produce uniform Ag nanowires. Copper chloride may be used as a catalyst in organic chlorination reactions. Used with palladium in a catalytic synthesis of 3-haloindoles via an annulation process.

Signal Word

Danger

Hazard Classifications

Acute Tox. 4 Dermal - Acute Tox. 4 Oral - Aquatic Acute 1 - Aquatic Chronic 2 - Eye Dam. 1 - Skin Irrit. 2

Storage Class Code

8A - Combustible corrosive hazardous materials

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Handbook of Copper Compounds and Applications null
Chemistry (Weinheim An Der Bergstrasse, Germany) null
Noble-Metal Nanostructures with Controlled Morphologies
Jiang M, et al.
Material Matters, 4(1) null
Synthesis, 1841-1841 (2007)
Pieter Spincemaille et al.
Toxicology and applied pharmacology, 280(2), 345-351 (2014-08-20)
Wilson disease (WD) is caused by accumulation of excess copper (Cu) due to a mutation in the gene encoding the liver Cu transporter ATP7B, and is characterized by acute liver failure or cirrhosis and neuronal cell death. We investigated the

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