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MAB1345

Sigma-Aldrich

Anti-Collagen Type VII Antibody, CT, clone LH7.2

ascites fluid, clone LH7.2, Chemicon®

Synonym(s):

Anti-EBD1, Anti-EBR1, Anti-NDNC8

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About This Item

UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41

biological source

mouse

Quality Level

100

antibody form

ascites fluid

antibody product type

primary antibodies

clone

LH7.2, monoclonal

species reactivity

human

manufacturer/tradename

Chemicon®

technique(s)

immunocytochemistry: suitable

isotype

IgG

NCBI accession no.

NM_000094.3

UniProt accession no.

Q02388

shipped in

dry ice

target post-translational modification

unmodified

Gene Information

human ... COL7A1(1294)

Specificity

Carboxy terminal peptide of type VII collagen.

Immunogen

Collagen type VII.

Application

Anti-Collagen Type VII Antibody, C-terminus, clone LH7.2 is an antibody against Collagen Type VII for use in IC.
Immunohistochemistry.

Optimal working dilutions must be determined by end user.

Physical form

Ascites. Liquid with 0.1% sodium azide.

Storage and Stability

Maintain at -20°C in convenient aliquots for up to 12 months. Avoid repeated freeze/thaw cycles.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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Storage Class

10 - Combustible liquids

wgk_germany

WGK 1

flash_point_f

Not applicable

flash_point_c

Not applicable

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Distribution of basement membrane zone components in bullous lesions of subepidermal blistering diseases.
Satomi Hosoda et al.
The Journal of dermatology, 40(3), 211-212 (2013-01-09)
Wakana Matsumura et al.
The Journal of investigative dermatology, 139(10), 2115-2124 (2019-05-06)
Inherited skin disorders have been reported recently to have sporadic normal-looking areas, where a portion of the keratinocytes have recovered from causative gene mutations (revertant mosaicism). We observed a case of recessive dystrophic epidermolysis bullosa treated with cultured epidermal autografts
Dimitra Kiritsi et al.
The Journal of clinical investigation, 122(5), 1742-1746 (2012-04-03)
Spontaneous gene repair, also called revertant mosaicism, has been documented in several genetic disorders involving organs that undergo self-regeneration, including the skin. Genetic reversion may occur through different mechanisms, and in a single individual, the mutation can be repaired in
Cristina Chamorro et al.
Molecular therapy. Nucleic acids, 5(4), e307-e307 (2016-04-06)
Clonal gene therapy protocols based on the precise manipulation of epidermal stem cells require highly efficient gene-editing molecular tools. We have combined adeno-associated virus (AAV)-mediated delivery of donor template DNA with transcription activator-like nucleases (TALE) expressed by adenoviral vectors to
High concordance between clinical diagnosis of epidermolysis bullosa and immunofluorescence with a small, well-matched antibody panel.
Irene Meester et al.
The Australasian journal of dermatology, 59(1), 73-76 (2017-07-15)
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