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SAB5600124

Sigma-Aldrich

Anti-Crotonyl-Histone H3 (Lys9) antibody, Rabbit monoclonal

recombinant, expressed in HEK 293 cells, clone RM339, purified immunoglobulin

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About This Item

UNSPSC Code:
12352203
NACRES:
NA.41

recombinant

expressed in HEK 293 cells

antibody form

purified immunoglobulin

antibody product type

primary antibodies

clone

RM339, monoclonal
recombinant monoclonal

form

buffered aqueous glycerol solution

species reactivity

vertebrates

isotype

IgG

UniProt accession no.

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

crotonylation (Lys9)

Gene Information

human ... H3C1(8350)

Specificity

This antibody reacts to Histone H3 crotonylated at Lysine 9 (K9cr), and does not cross-react with acetylated or butyrylated Lysine 9. No cross reactivity  with other crotonylated Lysines in histone H3

Immunogen

A crotonyl-peptide corresponding to Crotonyl-Histone H3 (Lys9).

Features and Benefits

Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.

Physical form

Solution in phosphate buffered saline containing 50% glycerol, 1% BSA and 0.09% sodium azide.

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Storage Class

10 - Combustible liquids

wgk_germany

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable


Certificates of Analysis (COA)

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Christopher McCrory et al.
mBio, 14(6), e0260523-e0260523 (2023-11-06)
Macrophages curtail the proliferation of the pathogen Candida albicans within human body niches. Within macrophages, C. albicans adapts its metabolism and switches to invasive hyphal morphology. These adaptations enable fungal growth and immune escape by triggering macrophage lysis. Transcriptional programs

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