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Key Documents

SAB4500120

Sigma-Aldrich

Anti-C/EBP-ε antibody produced in rabbit

affinity isolated antibody

Synonym(s):

C/EBP ε, CCAAT/enhancer binding protein ε, CEBPE

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100 μG
$392.00

$392.00

Usually ships in 5 business days. (Orders outside of US, please allow an additional 1-2 weeks for delivery)
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100 μG
$392.00

About This Item

UNSPSC Code:
12352203
NACRES:
NA.41

$392.00

Usually ships in 5 business days. (Orders outside of US, please allow an additional 1-2 weeks for delivery)

biological source

rabbit

Quality Level

100

conjugate

unconjugated

antibody form

affinity isolated antibody

antibody product type

primary antibodies

clone

polyclonal

form

buffered aqueous solution

mol wt

antigen 30 kDa

species reactivity

human

concentration

~1 mg/mL

technique(s)

ELISA: 1:1000
immunofluorescence: 1:100-1:500
immunohistochemistry: 1:50-1:100
western blot: 1:500-1:1000

NCBI accession no.

Q15744

UniProt accession no.

Q15744

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

unmodified

Gene Information

human ... CEBPE(1053)

General description

Anti-C/EBP-ε Antibody detects endogenous levels of total C/EBP-ε protein.

Immunogen

The antiserum was produced against synthesized peptide derived from human C/EBP-epsilon.

Immunogen Range: 40-89

Features and Benefits

Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.

Physical form

Rabbit IgG in phosphate buffered saline (without Mg2+ and Ca2+), pH 7.4, 150mM NaCl, 0.02% sodium azide and 50% glycerol.

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Storage Class

10 - Combustible liquids

wgk_germany

nwg

flash_point_f

Not applicable

flash_point_c

Not applicable

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Certificates of Analysis (COA)

Lot/Batch Number
210827

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Jie Sun et al.
Journal of cellular biochemistry, 119(3), 2891-2899 (2017-11-08)
Osteoarthritis (OA) is the leading degenerative joint disease and featured by articular cartilage destruction, where chondrocyte apoptosis plays a critical role. Semaphorin-3A (Sema3A) has been implicated in OA chondrocyte physiology. In this study we aimed to uncover how Sema3A signaling

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