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Key Documents

RAB1228

Sigma-Aldrich

Feline CXCL1 ELISA

for serum, plasma and cell culture supernatants

Synonym(s):

C-X-C motif chemokine 1, Growth-regulated alpha protein, Platelet-derived growth factor-inducible protein KC, Secretory protein N51

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$896.00

About This Item

UNSPSC Code:
41116158
NACRES:
NA.32

$896.00

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species reactivity

feline

technique(s)

ELISA: suitable

assay range

inter-assay cv: <12%
intra-assay cv: <10%

shipped in

wet ice

storage temp.

−20°C

General description

Chemokine (C-X-C motif) ligand 1 (CXCL1), a member of the CXC chemokine family,[1] is a small cytokine expressed by epithelial cells, macrophages, and neutrophils. It can stimulate progression, tumorigenesis, and recurrence in bladder cancer, pancreatic cancer, and glioma.[2] CXCL1 is capable of specifically binding to the G protein-coupled receptor chemokine (C-X-C motif) receptor 2 (CXCR2).[1] This ELISA antibody pair detects feline chemokine (C-X-C motif) ligand 1 (CXCL1).

Application

For research use only. Not for use in diagnostic procedures.
Please refer to the attached Protocolfor details.

Other Notes

A sample Certificate of Analysis is available for this product. Please type the word sample in the text box provided for lot number.

pictograms

Corrosion
GHS05

signalword

Warning

hcodes

H290

Hazard Classifications

Met. Corr. 1

Storage Class

8A - Combustible corrosive hazardous materials

flash_point_f

Not applicable

flash_point_c

Not applicable

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Certificates of Analysis (COA)

Lot/Batch Number
0308I0962
0315I0962
0622H0962

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Chengcheng Yang et al.
International journal of oncology, 55(3), 684-696 (2019-07-20)
Chemokine (C‑X‑C motif) ligand 1 (CXCL1), a member of the CXC chemokine family, has been reported to be a critical factor in inflammatory diseases and tumor progression; however, its functions and molecular mechanisms in estrogen receptor α (ER)‑negative breast cancer (BC) remain largely unknown. The
Wahafu Alafate et al.
CNS neuroscience & therapeutics, 26(4), 475-485 (2020-03-19)
Glioblastoma (GBM) is identified as a lethal malignant tumor derived from the nervous system. Despite the standard clinical strategy including maximum surgical resection, temozolomide (TMZ) chemotherapy, and radiotherapy, the median survival of GBM patients remains <15 months. Accumulating evidence indicates that

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