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T-500

Supelco

Testosterone CRM in Serum solution

ampule of 1 mL (Blank), certified reference material, Cerilliant®

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About This Item

Empirical Formula (Hill Notation):
C19H28O2
Molecular Weight:
288.42
UNSPSC Code:
12352200

grade

certified reference material

form

liquid

packaging

ampule of 1 mL (Blank)

manufacturer/tradename

Cerilliant®

technique(s)

gas chromatography (GC): suitable
liquid chromatography (LC): suitable

application(s)

clinical testing

format

single component solution

storage temp.

−20°C

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General description

Certified reference material of testosterone in stripped serum for clinical research applications by mass spectrometry. Individual levels of testosterone in stripped serum are offered separately at concentrations ranging from 2 to 2,000 ng/dL. Each individual level is ready to use as a linearity standard or for calibration verification in LC-MS/MS testosterone methods. Separate options for a blank and 13C- or deuterium labeled internal standards of testosterone are also available.

Legal Information

CERILLIANT is a registered trademark of Merck KGaA, Darmstadt, Germany

Storage Class

12 - Non Combustible Liquids

wgk_germany

WGK 1

flash_point_f

Not applicable

flash_point_c

Not applicable


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Severe acute pancreatitis (SAP) results in high mortality. This is partly because of early multiple organ dysfunction syndromes that are usually caused by systemic inflammatory response syndrome (SIRS). Many studies have reported the beneficial effects of emodin against SAP with
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Cyclooxygenases and lipoxygenases are proinflammatory enzymes; the former affects platelet aggregation, vasoconstriction, vasodilatation and later the development of atherosclerosis. Red wines from Georgia and central and western Europe inhibited cyclooxygenase-1 (COX-1) activity in the range of 63-94%, cyclooxygenase-2 (COX-2) activity
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PloS one, 8(1), e54195-e54195 (2013-01-18)
A(2A) adenosine receptors (ARs) play a key role in the inhibition of the inflammatory process. The purpose of this study was to evaluate the modulation of A(2A)ARs in rheumatoid arthritis (RA) patients after different pharmacological treatments and to investigate the

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