280968
N-(2-Mercaptopropionyl)glycine
99%
Synonym(s):
Tiopronin
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About This Item
Linear Formula:
CH3CH(SH)CONHCH2COOH
CAS Number:
Molecular Weight:
163.19
EC Number:
217-778-4
UNSPSC Code:
12352103
PubChem Substance ID:
Beilstein/REAXYS Number:
1859822
MDL number:
InChI key
YTGJWQPHMWSCST-UHFFFAOYSA-N
InChI
1S/C5H9NO3S/c1-3(10)5(9)6-2-4(7)8/h3,10H,2H2,1H3,(H,6,9)(H,7,8)
SMILES string
CC(S)C(=O)NCC(O)=O
assay
99%
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Jung Ah Lee et al.
Journal of Korean medical science, 27(5), 547-552 (2012-05-09)
We previously demonstrated that there are acute and delayed phases of renal protection against renal ischemia and reperfusion (IR) injury with renal ischemic preconditioning (IPC). This study assessed whether hepatic IPC could also reduce distant renal IR injury through the
Jing Xu et al.
Journal of biomedical materials research. Part A, 100(3), 738-745 (2012-01-04)
Hydroxyapatite (HA) nanoparticles have been reported to exhibit anti-tumor effects on various human cancers, but the effects of HA on glioma cells remain unclear. The aim of this study was to explore whether HA can inhibit the proliferation and induce
Christoph Becker-Pauly et al.
Molecular & cellular proteomics : MCP, 10(9), M111-M111 (2011-06-23)
Astacins are secreted and membrane-bound metalloproteases with clear associations to many important pathological and physiological processes. Yet with only a few substrates described their biological roles are enigmatic. Moreover, the lack of knowledge of astacin cleavage site specificities hampers assay
Yung-Kuo Lin et al.
Clinical science (London, England : 1979), 122(3), 121-132 (2011-09-02)
Ischaemia and reperfusion contribute to the genesis of AF (atrial fibrillation). PVs (pulmonary veins) and the atria are important foci for AF initiation and maintenance. However, the effect of ischaemia and reperfusion on PVs and the atria has not yet
Andrew S Goldsborough et al.
Journal of medicinal chemistry, 54(14), 4987-4997 (2011-06-11)
A major challenge in the treatment of cancer is multidrug resistance (MDR) that develops during chemotherapy. Here we demonstrate that tiopronin (1), a thiol-substituted N-propanoylglycine derivative, was selectively toxic to a series of cell lines expressing the drug efflux pump
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