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Merck
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Key Documents

AB15620

Sigma-Aldrich

Anti-Olig1 Antibody

Chemicon®, from rabbit

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About This Item

Code UNSPSC :
12352203
eCl@ss :
32160702
Nomenclature NACRES :
NA.41

Source biologique

rabbit

Niveau de qualité

Forme d'anticorps

purified antibody

Type de produit anticorps

primary antibodies

Clone

polyclonal

Espèces réactives

rat, mouse

Fabricant/nom de marque

Chemicon®

Technique(s)

immunocytochemistry: suitable
immunohistochemistry: suitable
immunoprecipitation (IP): suitable
western blot: suitable

Numéro d'accès NCBI

Numéro d'accès UniProt

Conditions d'expédition

wet ice

Modification post-traductionnelle de la cible

unmodified

Informations sur le gène

Spécificité

Olig-1. Recognizes the ~27 kDa Olig-1 protein by Western blot.

Immunogène

Recombinant Olig-1.

Application

Detect the Olig1 protein using this Anti-Olig1 validated for use in IP, WB, ICC & IHC.
Research Category
Neuroscience
Research Sub Category
Neuronal & Glial Markers
Western blot: 1:2,500-1:5,000 on rat and mouse.
Immunohistochemistry: 1:1,000-1:5,000
Immunocytochemistry: 1:1,000-1:5,000
Immunoprecipitation: 1:50-1:100

Optimal working dilutions must be determined by the end user.

Description de la cible

27 kDa

Forme physique

Format: Purified
Liquid in 100 mM Glycine, 200 mM Tris, pH 8.0.

Stockage et stabilité

Maintain at 2-8°C in undiluted aliquots for up to 6 months after date of receipt.

Informations légales

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Clause de non-responsabilité

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Code de la classe de stockage

12 - Non Combustible Liquids

Classe de danger pour l'eau (WGK)

WGK 1

Point d'éclair (°F)

Not applicable

Point d'éclair (°C)

Not applicable


Certificats d'analyse (COA)

Recherchez un Certificats d'analyse (COA) en saisissant le numéro de lot du produit. Les numéros de lot figurent sur l'étiquette du produit après les mots "Lot" ou "Batch".

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Retrouvez la documentation relative aux produits que vous avez récemment achetés dans la Bibliothèque de documents.

Consulter la Bibliothèque de documents

Huanjia Gao et al.
Frontiers in neuroscience, 18, 1408205-1408205 (2024-07-26)
Vascular dementia (VaD) is a complex neurodegenerative disorder. We previously found that treatment of VaD in middle-aged male rats subjected to multiple microinfarction (MMI) with AV-001, a Tie2 receptor agonist, significantly improves cognitive function. Age and sex affect the development
Activation of developmental nuclear fibroblast growth factor receptor 1 signaling and neurogenesis in adult brain by ?7 nicotinic receptor agonist.
Narla, ST; Klejbor, I; Birkaya, B; Lee, YW; Morys, J; Stachowiak, EK; Prokop et al.
Stem Cells Translational Medicine null
Kimberly L P Long et al.
Translational psychiatry, 11(1), 631-631 (2021-12-15)
Individual reactions to traumatic stress vary dramatically, yet the biological basis of this variation remains poorly understood. Recent studies demonstrate the surprising plasticity of oligodendrocytes and myelin with stress and experience, providing a potential mechanism by which trauma induces aberrant
Till Scheuer et al.
Developmental neurobiology, 79(3), 222-235 (2019-01-24)
In preterm infants, the changes from fetal life to ex-utero conditions often coincide with reduced growth and white matter damage of the cerebellum. The premature increase in arterial oxygen tension caused by preterm birth may dysregulate cerebellar development. In a
Kirill Shumilov et al.
Journal of neuroinflammation, 21(1), 171-171 (2024-07-16)
White matter injury (WMI) is thought to be a major contributor to long-term cognitive dysfunctions after traumatic brain injury (TBI). This damage occurs partly due to apoptotic death of oligodendrocyte lineage cells (OLCs) after the injury, triggered directly by the

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