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GF90165023

Copper - O.F.H.C.

sheet, 100x100mm, thickness 6.35mm, 99.95+%

Synonyme(s) :

Copper, CV003100

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About This Item

Formule empirique (notation de Hill):
Cu
Numéro CAS:
Poids moléculaire :
63.55
Numéro MDL:
Code UNSPSC :
12141711
ID de substance PubChem :
Nomenclature NACRES :
NA.23

Pureté

≥99.95%

Forme

sheet

Fabricant/nom de marque

Goodfellow 901-650-23

Résistivité

1.673 μΩ-cm, 20°C

Point d'ébullition

2567 °C (lit.)

Pf

1083.4 °C (lit.)

Densité

8.94 g/mL at 25 °C (lit.)

Chaîne SMILES 

[Cu]

InChI

1S/Cu

Clé InChI

RYGMFSIKBFXOCR-UHFFFAOYSA-N

Description générale

For updated SDS information please visit www.goodfellow.com.

Informations légales

Product of Goodfellow

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Consulter la Bibliothèque de documents

Alina Fedoseienko et al.
Annals of the New York Academy of Sciences, 1314, 6-14 (2014-04-05)
Copper is an important cofactor in numerous biological processes in all living organisms. However, excessive copper can be extremely toxic, so it is vital that the copper level within a cell is tightly regulated. The damaging effect of copper is
Adam Southon et al.
Metallomics : integrated biometal science, 5(10), 1346-1356 (2013-08-02)
Copper (Cu) is an essential redox active metal that is potentially toxic in excess. Multicellular organisms acquire Cu from the diet and must regulate uptake, storage, distribution and export of Cu at both the cellular and organismal levels. Systemic Cu
Giulia Cheloni et al.
Aquatic toxicology (Amsterdam, Netherlands), 155, 275-282 (2014-07-30)
The present study showed the important role of light intensity and spectral composition on Cu uptake and effects on green alga Chlamydomonas reinhardtii. High-intenisty light (HL) increased cellular Cu concentrations, but mitigated the Cu-induced decrease in chlorophyll fluorescence, oxidative stress
Lelita T Braiterman et al.
Proceedings of the National Academy of Sciences of the United States of America, 111(14), E1364-E1373 (2014-04-08)
Wilson disease (WD) is a monogenic autosomal-recessive disorder of copper accumulation that leads to liver failure and/or neurological deficits. WD is caused by mutations in ATP7B, a transporter that loads Cu(I) onto newly synthesized cupro-enzymes in the trans-Golgi network (TGN)
Nathalie Adam et al.
Environmental pollution (Barking, Essex : 1987), 194, 130-137 (2014-08-12)
In this study the uptake of ZnO and CuO nanoparticles by Daphnia magna was tested. Daphnids were exposed during 48 h to acute concentrations of the nanoparticles and corresponding metal salts. The Daphnia zinc and copper concentration was measured and the

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