Skip to Content
Merck
All Photos(1)

Documents

740950

Sigma-Aldrich

Solketal methacrylate

50 wt. % in dichloromethane, contains ~280 ppm 4-tert-butylcatechol as inhibitor

Synonym(s):

(2,2-Dimethyl-1,3-dioxolan-4-yl)methyl methacrylate, GMAK, Glycerine-1-methacrylate acetone ketal, SMA

Sign Into View Organizational & Contract Pricing
All Photos(1)

About This Item

Empirical Formula (Hill Notation):
C10H16O4
CAS Number:
7098-80-8
Molecular Weight:
200.23
MDL number:
MFCD21334174
UNSPSC Code:
12162002
PubChem Substance ID:
329765454
NACRES:
NA.23

form

liquid

contains

~280 ppm 4-tert-butylcatechol as inhibitor

concentration

50 wt. % in dichloromethane

refractive index

n20/D 1.438

density

1.153 g/mL at 25 °C

storage temp.

−20°C

SMILES string

CC(=C)C(=O)OCC1COC(C)(C)O1

InChI

1S/C10H16O4/c1-7(2)9(11)12-5-8-6-13-10(3,4)14-8/h8H,1,5-6H2,2-4H3

InChI key

JPFPDGRVRGETED-UHFFFAOYSA-N

Signal Word

Danger

Hazard Classifications

Carc. 2 - Eye Dam. 1 - Flam. Liq. 3 - Skin Irrit. 2 - STOT SE 3

Target Organs

Central nervous system, Respiratory system

Storage Class Code

3 - Flammable liquids

WGK

WGK 2

Flash Point(F)

81.0 °F

Flash Point(C)

27.2 °C

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

Already Own This Product?

Find documentation for the products that you have recently purchased in the Document Library.

Visit the Document Library

Frank Y Ma et al.
Nephrology (Carlton, Vic.), 24(9), 983-991 (2019-07-18)
Protease-activated receptor 2 (PAR2) has been implicated in the development of renal inflammation and fibrosis. In particular, activation of PAR2 in cultured tubular epithelial cells induces extracellular signal-regulated kinase signalling and secretion of fibronectin, C-C Motif Chemokine Ligand 2 (CCL2)
Hiroyasu Okazaki et al.
Experimental lung research, 45(7), 188-199 (2019-07-13)
Purpose/Aim of the Study: Wnt/β-catenin signaling was reported to be activated in pulmonary fibrosis, and was focused on as a target for antifibrotic therapy. However, the mechanism how the inhibition of Wnt/β-catenin signaling ameliorate pulmonary fibrosis has not been fully
Justin A Bishop et al.
The American journal of surgical pathology, 43(8), 1023-1032 (2019-05-17)
Salivary gland adenocarcinoma not otherwise specified (NOS) is a heterogenous group, likely containing distinct tumors not yet characterized. A growing number of low to intermediate-grade salivary carcinomas are now known to harbor tumor-specific gene fusions. On occasion, identifying a novel
Sarah L O'Beirne et al.
Journal of immunology (Baltimore, Md. : 1950), 195(6), 2788-2796 (2015-08-14)
Epithelial to mesenchymal cell transition (EMT), whereby fully differentiated epithelial cells transition to a mesenchymal phenotype, has been implicated in the pathogenesis of idiopathic pulmonary fibrosis (IPF). CXCR3 and its ligands are recognized to play a protective role in pulmonary
Rui Zheng et al.
The Journal of investigative dermatology, 139(9), 2029-2038 (2019-03-18)
After cutaneous injury, keratinocytes secrete paracrine factors that regulate wound cell functions; dysregulation of this signaling can lead to wound pathologies. Previously, we established that keratinocyte integrin α3β1 promotes wound angiogenesis through paracrine stimulation of endothelial cells. We hypothesize here
View All Related Papers

Our team of scientists has experience in all areas of research including Life Science, Material Science, Chemical Synthesis, Chromatography, Analytical and many others.

Contact Technical Service