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Key Documents

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28-3030

Sigma-Aldrich

Hidróxido de sodio solution

0.2 M

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About This Item

Fórmula lineal:
NaOH
Número de CAS:
1310-73-2
Peso molecular:
40.00
MDL number:
MFCD00003548
UNSPSC Code:
12352106
PubChem Substance ID:
329753134

vapor pressure

3 mmHg ( 37 °C)

form

liquid

availability

available only in Japan

concentration

0.2 M
1/5 N

density

1 g/cm3 at 20 °C

SMILES string

[OH-].[Na+]

InChI

1S/Na.H2O/h;1H2/q+1;/p-1

InChI key

HEMHJVSKTPXQMS-UHFFFAOYSA-M

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pictograms

Corrosion
GHS05

signalword

Warning

Hazard Classifications

Eye Irrit. 2 - Met. Corr. 1 - Skin Irrit. 2

Storage Class

8B - Non-combustible corrosive hazardous materials

wgk_germany

nwg

flash_point_f

Not applicable

flash_point_c

Not applicable

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Olof Idevall-Hagren et al.
The EMBO journal, 34(17), 2291-2305 (2015-07-24)
The extended synaptotagmins (E-Syts) are ER proteins that act as Ca(2+)-regulated tethers between the ER and the plasma membrane (PM) and have a putative role in lipid transport between the two membranes. Ca(2+) regulation of their tethering function, as well
Horacio Cardenas et al.
Epigenetics, 9(11), 1461-1472 (2014-12-04)
A key step in the process of metastasis is the epithelial-to-mesenchymal transition (EMT). We hypothesized that epigenetic mechanisms play a key role in EMT and to test this hypothesis we analyzed global and gene-specific changes in DNA methylation during TGF-β-induced
Aparajita H Chourasia et al.
EMBO reports, 16(9), 1145-1163 (2015-08-02)
BNip3 is a hypoxia-inducible protein that targets mitochondria for autophagosomal degradation. We report a novel tumor suppressor role for BNip3 in a clinically relevant mouse model of mammary tumorigenesis. BNip3 delays primary mammary tumor growth and progression by preventing the
Joshua S McLane et al.
Biophysical journal, 109(2), 249-264 (2015-07-23)
Mechanical properties of the tumor microenvironment have emerged as key factors in tumor progression. It has been proposed that increased tissue stiffness can transform stromal fibroblasts into carcinoma-associated fibroblasts. However, it is unclear whether the three to five times increase
Johannes Egerer et al.
The Journal of investigative dermatology, 135(10), 2368-2376 (2015-05-23)
Gerodermia osteodysplastica is a hereditary segmental progeroid disorder affecting skin, connective tissues, and bone that is caused by loss-of-function mutations in GORAB. The golgin, RAB6-interacting (GORAB) protein localizes to the Golgi apparatus and interacts with the small GTPase RAB6. In
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