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EMU003111

Sigma-Aldrich

MISSION® esiRNA

targeting mouse Ihh

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About This Item

UNSPSC Code:
41105324
NACRES:
NA.51

description

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product line

MISSION®

form

lyophilized powder

esiRNA cDNA target sequence

CTCTAACCACTGCCCTCCTGGAACTGCTGTGCTGGATCCAAAGGCCTCCTCACCAGGAAGGCTCTGGCCCTGGAAGGCACCTGGCCTGAGGTTGTCTCCGTCCTCTGTGCCAGAGTGGAGACACCATTGAGACTTGACCAGGTTTGCTGGGCCCCGAACCTTCATCTTGGTGTAGAGCTGTGAACTGAGCTGACAAGCGTGTGGTAGGCTCTCTTTTCCTAGAGACCGTAAGACCCAGCTAGCTCTGGCTGCGATTCTTCACACGCATTCCATCTGTCTTTGGACTGCTTACTCCAATGTTTCTCGGGGCCTGGGATTGTGACTTTACTGTTGGCAACTGATCACAGTATGAAGAGAGGCTGCCCGTAGATGGGCTTGCACCTCAGTCGATGCTGCTAGATTCCC

Ensembl | mouse accession no.

NCBI accession no.

shipped in

ambient

storage temp.

−20°C

Gene Information

General description

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Legal Information

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

Storage Class Code

10 - Combustible liquids

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable


Certificates of Analysis (COA)

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Ang Deng et al.
Experimental and therapeutic medicine, 15(1), 789-794 (2018-02-13)
The proliferative rate of chondrocytes affects bone elongation. Chondrocyte hypertrophy is required for endochondral bone formation as chondrocytes secrete factors required for osteoblast differentiation and maturation. Previous studies have demonstrated that the Indian hedgehog (Ihh) signaling pathway is a key
Shaowei Wang et al.
European spine journal : official publication of the European Spine Society, the European Spinal Deformity Society, and the European Section of the Cervical Spine Research Society, 24(8), 1720-1728 (2015-05-11)
To determine the role of Indian hedgehog (Ihh) signaling in human cartilage endplate (CEP) degeneration. CEP-degenerated tissues from patients with Modic I or II changes (n = 9 and 45, respectively) and normal tissues from vertebral burst fracture patients (n

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