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Key Documents

E8029

Sigma-Aldrich

E-Toxate Endotoxin standard

for endotoxin quantitation

Synonym(s):

E. coli 0.55:B5 LPS standard

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About This Item

UNSPSC Code:
12352202
NACRES:
NA.24

Quality Level

contains

solubilizer

storage temp.

2-8°C

Application

E-Toxate endotoxin standard was used along with the limulus amebocyte lysate assay kit to determine the concentration of endotoxins in isolated samples of human holotransferrin and apotransferrin used to treat primary hepatocyte cultures.

Other Notes

A preparation of E. coli 055:B5 lipopolysaccharide
Can be stored at 2-8 °C when stored separately from the E-Toxate kit.

Analysis Note

Standardized against USP reference standard endotoxin (RSE)

Legal Information

E-Toxate is a trademark of Sigma-Aldrich Co. LLC

Storage Class Code

12 - Non Combustible Liquids

WGK

WGK 2

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Anna Scanu et al.
International journal of molecular sciences, 23(21) (2022-11-12)
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Kruppel-like factor 2 (KLF2) has been linked with fibrosis and neutrophil-associated thromboinflammation; however, its role in COVID-19 remains elusive. We investigated the effect of disease microenvironment on the fibrotic potential of human lung fibroblasts (LFs) and its association with KLF2
Lan Lin et al.
Blood, 110(6), 2182-2189 (2007-06-02)
The peptide hormone hepcidin is the principal regulator of systemic iron homeostasis. We examined the pathway by which iron stimulates the production of hepcidin. In humans who ingested 65 mg of iron, the increase in transferrin saturation preceded by hours
Akrivi Chrysanthopoulou et al.
JCI insight, 6(18) (2021-07-30)
Innate immunity and chronic inflammation are involved in atherosclerosis and atherothrombosis, leading to target organ damage in essential hypertension (EH). However, the role of neutrophils in EH is still elusive. We investigated the association between angiotensin II (Ang II) and

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