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EMU072881

Sigma-Aldrich

MISSION® esiRNA

targeting mouse 1190002h23rik

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About This Item

UNSPSC Code:
41105324
NACRES:
NA.51

description

Powered by Eupheria Biotech

product line

MISSION®

form

lyophilized powder

esiRNA cDNA target sequence

AGCGCCACTTCCACTATGAGGAGCACCTAGAGCGCATGAAGCGGCGCAGCAGCGCCAGCATCAGCAACAGCAGCGGCTTCAGCGACTCGGAGAGTGCAGACTCAGTGTACAGGGACAGCTTCACCTTCAGTGATGAGAAGCTGAATTCTCCAACCAACTCCTCTCCAGCTCTCCTGCCCTCCGCTGTCACTCCTCGGAAAGCCAAATTAGGTGACACTAAAGAGCTCGAAGACTTCATTGCCGATCTGGACAGGACCTTAGCAAGTATGTGAAGCAAGGAGTTTGGGGTCCAGAAGGCTCCGAGGACCTGGCAAATCGGCTACTAGAATCTGCTGTGGAAGAGAGCAGAGCTAAGACTCCTGCCCCCTGACCATTCTTAGTTCACTATAACATTAGCCATTGGGCCCATCTCTGGGCAGTTCGGAGAGTGAAGCT

Ensembl | mouse accession no.

NCBI accession no.

shipped in

ambient

storage temp.

−20°C

Gene Information

General description

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Legal Information

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

Storage Class

10 - Combustible liquids

flash_point_f

Not applicable

flash_point_c

Not applicable


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ACTRIMS-ECTRIMS MSBoston 2014: Poster Sessions 2.
Multiple sclerosis (Houndmills, Basingstoke, England), 20(1 Suppl), 285-496 (2014-09-11)
Ran Xu et al.
Molecular and cellular biochemistry, 394(1-2), 109-118 (2014-05-17)
Response gene to complement 32 (RGC32) is a novel protein originally identified as a cell cycle activator and has been demonstrated to be overexpressed in a variety of human malignancies, including lung cancer. However, the potential role of RGC32 in
Peng Zhao et al.
Cellular & molecular immunology, 12(6), 692-699 (2014-11-25)
Response gene to complement 32 (RGC-32) is a cell cycle regulator involved in the proliferation, differentiation and migration of cells and has also been implicated in angiogenesis. Here we show that RGC-32 expression in macrophages is induced by IL-4 and

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