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D0513

Supelco

Dibucaine

analytical standard

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About This Item

Fórmula empírica (notación de Hill):
C20H29N3O2
Número de CAS:
85-79-0
Peso molecular:
343.46
EC Number:
201-632-1
MDL number:
MFCD00047595
UNSPSC Code:
41116107
PubChem Substance ID:
24893238

grade

analytical standard

technique(s)

HPLC: suitable
gas chromatography (GC): suitable

application(s)

cleaning products
cosmetics
food and beverages
forensics and toxicology
personal care
pharmaceutical (small molecule)
veterinary

format

neat

SMILES string

CCCCOc1cc(C(=O)NCCN(CC)CC)c2ccccc2n1

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Application

Refer to the product′s Certificate of Analysis for more information on a suitable instrument technique. Contact Technical Service for further support.

Biochem/physiol Actions

Potent, long-acting local anesthetic

pictograms

Environment
GHS09

hcodes

H411

Hazard Classifications

Aquatic Chronic 2

Storage Class

11 - Combustible Solids

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

dust mask type N95 (US), Eyeshields, Faceshields, Gloves

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Certificados de análisis (COA)

Lot/Batch Number
049K1165
067K1134
066K1236
129H1011
086H1018

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Bryan Heit et al.
American journal of physiology. Cell physiology, 300(1), C33-C41 (2010-10-12)
Electrostatic interactions with negative lipids contribute to the subcellular localization of polycationic proteins. In situ measurements using cytosolic probes of surface charge indicate that normal mitochondria are not noticeably electronegative. However, during apoptosis mitochondria accrue negative charge and acquire the
Ana Peropadre et al.
Chemical research in toxicology, 24(2), 185-192 (2011-01-26)
A wide range of cationic amphiphilic drugs (CADs) from different therapeutic areas are known to cause phospholipidosis both in vivo and in vitro. Although the relevance of this storage disorder for human health remains uncertain, CADs have been repeatedly associated
V Arizza et al.
Fish & shellfish immunology, 30(4-5), 1014-1023 (2011-02-04)
Hemocytes from the ascidian Ciona intestinalis exert in vitro Ca²+-dependent cytotoxic activity toward mammalian erythrocytes and K562 cells. To examine the lytic mechanism, hemocyte populations were separated (B1-B6 bands) through a Percoll discontinuous density gradient, the hemocyte cytotoxic activity (HCA)
W Christopher Risher et al.
PloS one, 6(7), e22351-e22351 (2011-07-27)
Spreading depolarizations that occur in patients with malignant stroke, subarachnoid/intracranial hemorrhage, and traumatic brain injury are known to facilitate neuronal damage in metabolically compromised brain tissue. The dramatic failure of brain ion homeostasis caused by propagating spreading depolarizations results in
Heather A Douglas et al.
Journal of neurophysiology, 105(4), 1482-1494 (2011-01-29)
Recurring waves of peri-infarct depolarizations (PIDs) propagate across gray matter in the hours and days following stroke, expanding the primary site of injury. Ischemic depolarization (termed anoxic depolarization or AD in live brain slices) is PID-like but immediately arises in
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