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Sigma-Aldrich

Aluminum chloride solution

1.0 M in nitrobenzene

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About This Item

Empirical Formula (Hill Notation):
AlCl3
CAS Number:
7446-70-0
Molecular Weight:
133.34
MDL number:
MFCD00003422
UNSPSC Code:
12352302
PubChem Substance ID:
329752764
NACRES:
NA.55

form

liquid

reaction suitability

reagent type: catalyst
core: aluminum

concentration

0.95-1.10 M (EDTA titration)
1.0 M in nitrobenzene

density

1.249 g/mL at 25 °C

SMILES string

Cl[Al](Cl)Cl

InChI

1S/Al.3ClH/h;3*1H/q+3;;;/p-3

InChI key

VSCWAEJMTAWNJL-UHFFFAOYSA-K

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signalword

Danger

Hazard Classifications

Acute Tox. 3 Dermal - Acute Tox. 3 Inhalation - Acute Tox. 3 Oral - Aquatic Chronic 3 - Carc. 2 - Eye Dam. 1 - Repr. 1B - Skin Corr. 1B - STOT RE 1 Inhalation

target_organs

Blood

supp_hazards

EUH014,EUH071

Storage Class

6.1A - Combustible acute toxic Cat. 1 and 2 / very toxic hazardous materials

wgk_germany

WGK 3

flash_point_f

190.4 °F - closed cup

flash_point_c

88 °C - closed cup

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Acceleration of the Diels-Alder reaction by aluminum chloride.
Yates P and Eaton P.
Journal of the American Chemical Society, 82(16), 4426-4437 (1960)
Zhiliang Huang et al.
Organic & biomolecular chemistry, 11(11), 1810-1814 (2013-02-08)
AlCl(3) promoted Friedel-Crafts acylation between 4-tert-butylbenzoyl chloride and mesitylene was investigated. The donor-acceptor complex was observed as the major species. Kinetic investigation demonstrated that the reaction was first-order on the donor-acceptor complex and zero-order on ArH, suggesting that the donor-acceptor
Amination of< i> exo</i>-2-chloronorbornane and norbornane with trichloramine-aluminum chloride.
Kovacic P, et al.
Tetrahedron, 26(2), 529-538 (1970)
Diagnosis and management of hyperhidrosis.
R A Benson et al.
BMJ (Clinical research ed.), 347, f6800-f6800 (2013-11-28)
Dharmalingam Prakash et al.
Neuromolecular medicine, 15(1), 192-208 (2013-01-15)
Aluminum (Al) is an environmental neurotoxin that affects cerebral functions and causes health complications. However, the role of Al in arbitrating glia homeostasis and pathophysiology remains obscure. Astrocyte, microglia activation (reactive gliosis), and associated inflammatory events play a decisive role
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