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860585P

Avanti

17:0 SM (d18:1/17:0)

Name: 17:0 SM (d18:1/17:0)
Brand: AVANTI

Avanti Research^™ - A Croda Brand 860585P, powder

Synonym(s):

N-heptadecanoyl-D-erythro-sphingosylphosphorylcholine

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About This Item

Empirical Formula (Hill Notation):

C₄₀H₈₁N₂O₆P

CAS Number:

121999-64-2

Molecular Weight:

717.05

UNSPSC Code:

12352211

NACRES:

NA.25

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Sigma-Aldrich

C8667

Cholesterol

Sigma-Aldrich

P0537

1,2-Dilinoleoyl-sn-glycero-3-phosphocholine

form

powder

packaging

pkg of 1 × 10 mg (860585P-10mg)
pkg of 1 × 5 mg (860585P-5mg)

manufacturer/tradename

Avanti Research^™ - A Croda Brand 860585P

lipid type

sphingolipids

shipped in

dry ice

storage temp.

−20°C

SMILES string

[H][C@](/C=C/CCCCCCCCCCCCC)(O)[C@@]([H])(NC(CCCCCCCCCCCCCCCC)=O)COP([O-])(OCC[N+](C)(C)C)=O

InChI

1S/C40H81N2O6P/c1-6-8-10-12-14-16-18-20-22-24-26-28-30-32-34-40(44)41-38(37-48-49(45,46)47-36-35-42(3,4)5)39(43)33-31-29-27-25-23-21-19-17-15-13-11-9-7-2/h31,33,38-39,43H,6-30,32,34-37H2,1-5H3,(H-,41,44,45,46)/b33-31+/t38-,39+/m0/s1

InChI key

YMQZQHIESOAPQH-JXGHDCMNSA-N

Application

N-heptadecanoyl-D-erythro-sphingosylphosphorylcholine (17:0 SM (d18:1/17:0)) has been used as an internal standard for the quantification of C17-sphingomyelin (SM) using mass spectrometry (MS). It may be used as an internal standard for the quantification of lipids in serum and skeletal muscles using liquid chromatography-mass spectrometry (LC-MS) analysis. It may also be used as an internal standard for retention time calibration and for the extraction of lipids from blood samples using methanol and methyl tert-butyl ether (MTBE).

Packaging

5 mL Amber Glass Screw Cap Vial (860585P-10mg)

5 mL Amber Glass Screw Cap Vial (860585P-5mg)

Legal Information

Avanti Research is a trademark of Avanti Polar Lipids, LLC

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Product No.

Description

Pricing

Storage Class

11 - Combustible Solids

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Deep Profiling of Immunosuppressive Glycosphingolipids and Sphingomyelins in Wild Cordyceps.

Jianing Mi et al.

Journal of agricultural and food chemistry, 66(34), 8991-8998 (2018-07-31)

Deep profiling of glycosphingolipids and sphingomyelins in wild Cordyceps was carried out by using offline chromatographic enrichment followed by ultrahigh performance liquid chromatography-ultrahigh definition-quadrupole time-of-flight mass spectrometry (UHPLC-UHD-Q-TOF-MS). A total of 119 glycosphingolipids (72 new ones) and 87 sphingomyelins (43

Long-term use of fluoxetine accelerates bone loss through the disruption of sphingolipids metabolism in bone marrow adipose tissue.

Huili Zhang et al.

Translational psychiatry, 10(1), 138-138 (2020-05-14)

Fluoxetine is a commonly prescribed antidepressant, and the mechanisms of increased bone fragility with its long-term use remain largely unknown. Here, we show that long-term administration of fluoxetine induces the disruption of sphingolipids metabolism in bone marrow adipose tissue (BMAT)through

Interventional effects of the direct application of "Sanse powder" on knee osteoarthritis in rats as determined from lipidomics via UPLC-Q-Exactive Orbitrap MS.

Peng Wu et al.

Chinese medicine, 15, 9-9 (2020-01-31)

Our previous clinical evidence suggested that the direct application of "Sanse powder" the main ingredient of "Yiceng" might represent an alternative treatment for knee osteoarthritis. However, the mechanism underlying its effect is poorly understood. In this study, we investigated the

Generation and quality control of lipidomics data for the alzheimer's disease neuroimaging initiative cohort.

Dinesh Kumar Barupal et al.

Scientific data, 5, 180263-180263 (2018-11-21)

Alzheimer's disease (AD) is a major public health priority with a large socioeconomic burden and complex etiology. The Alzheimer Disease Metabolomics Consortium (ADMC) and the Alzheimer Disease Neuroimaging Initiative (ADNI) aim to gain new biological insights in the disease etiology.

Skeletal muscle ceramides and relationship with insulin sensitivity after 2 weeks of simulated sedentary behaviour and recovery in healthy older adults.

Paul T Reidy et al.

The Journal of physiology, 596(21), 5217-5236 (2018-09-09)

Insulin sensitivity (as determined by a hyperinsulinaemic-euglyceamic clamp) decreased 15% after reduced activity. Despite not fully returning to baseline physical activity levels, insulin sensitivity unexpectedly, rebounded above that recorded before 2 weeks of reduced physical activity by 14% after the recovery

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