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Key Documents

AB1565

Sigma-Aldrich

Anti-Vasopressin Antibody

serum, Chemicon®

Synonym(s):

Anti-ADH, Anti-ARVP, Anti-AVP-NPII, Anti-AVRP, Anti-VP

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About This Item

UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41

biological source

rabbit

Quality Level

antibody form

serum

antibody product type

primary antibodies

clone

polyclonal

species reactivity

human, sheep, rat, mouse, rabbit

manufacturer/tradename

Chemicon®

technique(s)

immunocytochemistry: suitable
immunohistochemistry: suitable (paraffin)
radioimmunoassay: suitable
western blot: suitable

NCBI accession no.

UniProt accession no.

shipped in

dry ice

target post-translational modification

unmodified

Gene Information

human ... AVP(551)

Specificity

Recognizes Vasopressin. Less than 1% cross-reactivity with oxytocin.

Immunogen

Arginine vasopressin (Sigma) conjugated to thyroglobulin.

Application

Anti-Vasopressin Antibody detects level of Vasopressin & has been published & validated for use in IC, IH(P), RIA & WB.
Immunohistochemistry: 1:5,000-1:10,000 (18-24 hour incubation) on formalin, Bouin′s or Zamboni′s fixed tissue. Will react on paraffin embedded tissue.

Immunocytochemistry: 1:1,000-1:200,000 (18-24 hour incubation) on fetal rat neurons.

Immunoblotting: 1:1,000

RIA: 1:30,000-1:750,000. Sensitivity of <1 pg/mL.

Optimal working dilutions must be determined by the end user.
Research Category
Neuroscience
Research Sub Category
CNS Control of Metabolism

Hormones & Receptors

Linkage

Replaces: AB937

Physical form

Neat rabbit antisera. Liquid, with 0.1% sodium azide.
Unpurified

Storage and Stability

Maintain for 2 years at -20°C from date of shipment. Aliquot to avoid repeated freezing and thawing. For maximum recovery of product, centrifuge the original vial after thawing and prior to removing the cap.

Analysis Note

Control
Hypothalamus

Other Notes

Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Storage Class Code

10 - Combustible liquids

WGK

WGK 1


Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Sabine Michaëlle Duplan et al.
The European journal of neuroscience, 30(12), 2239-2249 (2010-01-23)
The bHLH-PAS transcription SIM1 is required for the development of all neurons of the paraventricular nucleus (PVN) and supraoptic nucleus (SON) of the hypothalamus. Mice with a loss of Sim1 die within a few days of birth, presumably because of
Tumour-specific arginine vasopressin promoter activation in small-cell lung cancer.
Coulson, JM; Stanley, J; Woll, PJ
British Journal of Cancer null
Jing Zhu et al.
Neuromodulation : journal of the International Neuromodulation Society, 19(5), 498-506 (2015-11-18)
The study aims to know the effect of electroacupuncture (EA) in maintenance of the homeostasis of the neuroendocrine system in hepatectomy rats and the involvement of arginine vasopressin (AVP) signaling in hypothalamus after EA was observed. Rats were randomly assigned
Carlos Dombret et al.
Human molecular genetics, 21(21), 4703-4717 (2012-08-07)
MAGED1, NECDIN and MAGEL2 are members of the MAGE gene family. The latter two of these genes have been involved in Prader-Willi syndrome (PWS), which includes hyperphagia, repetitive and compulsive behaviors, and cognitive impairment. Here, we show that Maged1-deficient mice
Vanessa A Jimenez et al.
Frontiers in cellular neuroscience, 9, 260-260 (2015-08-04)
A bidirectional relationship between stress and ethanol exists whereby stressful events are comorbid with problematic ethanol use and prolonged ethanol exposure results in adaptations of the physiological stress response. Endocrine response to stress is initiated in the hypothalamic paraventricular nucleus

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