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SMB01004

Sigma-Aldrich

Chrysogine

≥90% (LC/MS-ELSD)

Synonym(s):

(+)-Chrysogine, 2-[(1R)-1-hydroxyethyl]-3H-quinazolin-4-one

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About This Item

Empirical Formula (Hill Notation):
C10H10N2O2
CAS Number:
18326-30-2
Molecular Weight:
190.20
MDL number:
MFCD28011230
UNSPSC Code:
12352205
NACRES:
NA.25

biological source

plant

Assay

≥90% (LC/MS-ELSD)

form

solid

mol wt

190.2

application(s)

metabolomics
vitamins, nutraceuticals, and natural products

storage temp.

−20°C

InChI

1S/C10H10N2O2/c1-6(13)9-11-8-5-3-2-4-7(8)10(14)12-9/h2-6,13H,1H3,(H,11,12,14)

InChI key

BMBSGGZMJQTQSO-UHFFFAOYSA-N

Related Categories

General description

Chrysogine, a quinazolinone metabolite, is a yellow pigment commonly produced by Fusarium sambucinum, Penicillium chrysogenum, Penicillium notatum, and other filamentous fungi. Existing research indicates that this bioactive natural compound, exhibits various biological properties, including antibacterial, antifungal, and antiviral activities.

Application

It is a natural product derived from plant source that finds application in compound screening libraries, metabolomics, phytochemical, and pharmaceutical research.

Features and Benefits

  • High quality compound suitable for multiple research applications
  • Compatible with HPLC and mass spectrometry techniques

Other Notes

For additional information on our range of Biochemicals, please complete this form.

Storage Class Code

11 - Combustible Solids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Annarita Viggiano et al.
Applied and environmental microbiology, 84(4) (2017-12-03)
Chrysogine is a yellow pigment produced by Penicillium chrysogenum and other filamentous fungi. Although the pigment was first isolated in 1973, its biosynthetic pathway has so far not been resolved. Here, we show that deletion of the highly expressed nonribosomal

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