GF86610539
Manganese
foil, not light tested, 50x50mm, thickness 0.0125mm, permanent polyester support, 98.7%
Synonym(s):
Manganese, MN000160
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About This Item
Assay
≥98.7%
form
foil
manufacturer/tradename
Goodfellow 866-105-39
resistivity
185 μΩ-cm, 20°C
bp
1962 °C (lit.)
mp
1244 °C (lit.)
density
7.3 g/mL at 25 °C (lit.)
SMILES string
[Mn]
InChI
1S/Mn
InChI key
PWHULOQIROXLJO-UHFFFAOYSA-N
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General description
For updated SDS information please visit www.goodfellow.com.
Legal Information
Product of Goodfellow
Storage Class Code
13 - Non Combustible Solids
WGK
WGK 2
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
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Neurotoxicology, 36, 1-9 (2013-02-20)
Results from a meta-analysis of aggregated data provoked a new analysis using individual data on the neuropsychological performance of occupationally exposed workers. Data from eight studies examining 579 exposed and 433 reference participants were included, 28 performance variables analyzed. The
Biochimica et biophysica acta, 1837(8), 1257-1262 (2014-04-15)
The binding affinity of the two substrate-water molecules to the water-oxidizing Mn₄CaO₅ catalyst in photosystem II core complexes of the extremophilic red alga Cyanidioschyzon merolae was studied in the S₂ and S₃ states by the exchange of bound ¹⁶O-substrate against
Free radical biology & medicine, 62, 65-75 (2013-02-12)
Manganese (Mn) is an essential dietary nutrient, but an excess or accumulation can be toxic. Disease states, such as manganism, are associated with overexposure or accumulation of Mn and are due to the production of reactive oxygen species, free radicals
Stroke, 46(6), 1626-1632 (2015-04-25)
Nuclear erythroid 2 related factor 2 (Nrf2) is an astrocyte-enriched transcription factor that has previously been shown to upregulate cellular antioxidant systems in response to ischemia. Although resveratrol preconditioning (RPC) has emerged as a potential neuroprotective therapy, the involvement of
Annals of neurology, 75(4), 608-612 (2014-03-07)
We followed a patient with manganese transporter deficiency due to homozygous SLC30A10 mutations from age 14 years until his death at age 38 years and present the first postmortem findings of this disorder. The basal ganglia showed neuronal loss, rhodanine-positive
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