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S5007

Sigma-Aldrich

Sunflower seed oil

from Helianthus annuus, liquid, emollient

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About This Item

CAS Number:
EC Number:
MDL number:
UNSPSC Code:
12352200
NACRES:
NA.77

product name

Sunflower seed oil from Helianthus annuus,

density

0.92 g/mL at 25 °C (lit.)

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General description

Sunflower seed oil from Helianthus annuus contains saturated fatty acids (palmitic and stearic acids), monounsaturated fatty acid (oleic acid), and polyunsaturated acid (mostly linoleic acid). In addition, this edible oil also contains nonglyceridic contents including tocopherols and plant sterols, and is rich in vitamins and minerals.

Application

Sunflower seed oil from Helianthus annuus was used as a vehicle to administer tamoxifen and 4-hydroxytamoxifen to mice.

Biochem/physiol Actions

Sunflower seed oil is one of the most used vegetable oils in human nutrition. Oilseed crop sunflower (Helianthus annuus) is also used for biodiesel production. Sunflower oil is used as an emollient in cosmetics, and it serves as an effective phenolic antioxidant. Phytosterols in sunflower oil help in regulating the cholesterol levels in the body. Therefore, sunflower oil may be used to treat pathological conditions such as like acne, arthritis, and hair damage.

Supplementary Hazards

Storage Class Code

10 - Combustible liquids

WGK

WGK 3

Flash Point(F)

>230.0 °F

Flash Point(C)

> 110 °C

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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PloS one, 14(7), e0220048-e0220048 (2019-07-26)
Microbial surface properties are important for interactions with the environment in which cells reside. Surface properties of lactic acid bacteria significantly vary and some strains can form strong emulsions when mixed with a hydrocarbon. Lactococcus lactis NCDO712 forms oil-in-water emulsions
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Accumulation of senescent cells over time contributes to aging and age-related diseases. However, what drives senescence in vivo is not clear. Here we used a genetic approach to determine if spontaneous nuclear DNA damage is sufficient to initiate senescence in

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