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一般說明
绵羊血清是从正常供体群中获取的混合制剂。
應用
绵羊血清可在免疫测定中用作阻断剂和对照品。它也被用于阻断冷冻大鼠唇上提肌组织切片的免疫组化非特异性反应。
绵羊血清已被用作免疫测定的阻断剂和对照品。
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訊號詞
Warning
危險分類
Aquatic Chronic 2 - Eye Irrit. 2 - Skin Irrit. 2 - Skin Sens. 1
儲存類別代碼
12 - Non Combustible Liquids
水污染物質分類（WGK）
WGK 3
閃點（°F）
Not applicable
閃點（°C）
Not applicable
個人防護裝備
Eyeshields, Gloves
其他客户在看
Emilia Evgenieva et al.
Neurorehabilitation and neural repair, 22(6), 754-768 (2008-07-10)
Using the rat facial nerve axotomy model, the authors recently showed that manual stimulation of denervated whiskerpad muscles reduced the posttransectional polyinnervation at the neuromuscular junctions and promoted full recovery of vibrissal whisking. Prompted by implications for rehabilitation therapy, the
A S Popratiloff et al.
The Journal of comparative neurology, 433(3), 364-379 (2001-04-12)
Chewing, swallowing, breathing, and vocalization in mammals require precise coordination of tongue movements with concomitant activities of the mimetic muscles. The neuroanatomic basis for this oro-facial coordination is not yet fully understood. After the stereotaxic microinjection of retrograde and anterograde
Stoyan P Pavlov et al.
Experimental neurology, 211(1), 292-300 (2008-04-03)
We have recently shown in rat that daily manual stimulation (MS) of vibrissal muscles promotes recovery of whisking and reduces polyinnervation of muscle fibers following repair of the facial nerve (facial-facial anastomosis, FFA). Here, we examined whether these positive effects
Ann Ng et al.
Developmental biology, 335(1), 208-215 (2009-09-08)
Glypican-3 (Gpc3) is a heparan sulfate proteoglycan (HSPG) expressed widely during vertebrate development. Loss-of-function mutations cause Simpson-Golabi-Behmel syndrome (SGBS), a rare and complex congenital overgrowth syndrome with a number of associated developmental abnormalities including congenital heart disease. We found that
Chang-Wen Hsieh et al.
The Journal of nutrition, 139(12), 2257-2265 (2009-10-09)
Obesity and type 2 diabetes are growing problems worldwide in adults and children. In this study, we focused on understanding the patterning of insulin resistance as a result of altered perinatal nutrition. We analyzed mice in which the binding site
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