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Merck
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Key Documents

RAB1027

Sigma-Aldrich

人CD59 ELISA试剂盒

for cell culture supernatants, plasma, and serum samples

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About This Item

分類程式碼代碼:
41116158
NACRES:
NA.32

物種活性

human

包裝

kit of 96 wells (12 strips x 8 wells)

技術

ELISA: suitable

輸入

sample type cell culture supernatant(s)
sample type serum sample(s)
sample type plasma

assay range

inter-assay cv: <12%
intra-assay cv: <10%
sensitivity: 40 pg/ml

檢測方法

colorimetric

運輸包裝

wet ice

儲存溫度

−20°C

基因資訊

human ... CD59(966)

一般說明

This ELISA antibody pair detects human CD59. Other species are not determined.

應用

For research use only. Not for use in diagnostic procedures.
Please refer to the attached Protocolfor details.

其他說明

A sample Certificate of Analysis is available for this product. Please type the word sample in the text box provided for lot number.

象形圖

Corrosion

訊號詞

Warning

危險聲明

防範說明

危險分類

Met. Corr. 1

儲存類別代碼

8A - Combustible corrosive hazardous materials

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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Sheng-Wei Luo et al.
Fish & shellfish immunology, 89, 486-497 (2019-04-14)
CD59, a multifunctional glycoprotein, not only plays a regulatory role in complement cascades, but also participates in modulation of teleostean immunity. In this study, full length sequence of EcCD59 was obtained, comprising a 5'UTR of 163 bp, an ORF of
Yang Yie Sio et al.
Scientific reports, 10(1), 715-715 (2020-01-22)
Post-glycosylphosphatidylinositol (GPI) attachment to proteins 3, also known as PGAP3 or PERLD1 (PER1-like domain-containing protein 1), participates in the lipid remodeling process of glycosylphosphatidylinositol (GPI) anchor proteins during post-translational modification. Functional defect in PERLD1 was previously hypothesized to influence this
Diego Martínez-López et al.
Journal of the American College of Cardiology, 75(16), 1926-1941 (2020-04-25)
The mechanisms underlying early atherosclerotic plaque formation are not completely understood. Moreover, plasma biomarkers of subclinical atherosclerosis are lacking. The purpose of this study was to analyze the temporal and topologically resolved protein changes taking place in human aortas with

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