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Merck
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主要文件

EMU039841

Sigma-Aldrich

MISSION® esiRNA

targeting mouse Runx1

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About This Item

UNSPSC代码:
41105324
NACRES:
NA.51

描述

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产品线

MISSION®

表单

lyophilized powder

esiRNA cDNA靶序列

TGATCCTATCCAGCCTTCCAACTACTGAAGCTGATTTTCAAGGCTACTTAAAAAAAAAAAAACTGCAGCAAACATTAATGGATTTCTGTTGTGTTTAAATTCTCTACAGATTGTATTGTAAATATTTTATGAAGTAGATCATATGTATATATTTATATATACGAGCACATACATTAGTAGCACAATCTTTTAAAGTTACGGCTCTTGCTTTTGAGAAACGAAGTGAGTTTTTCATGGTAAGAGGGGCGCTCTGTATGGAAGACACTCCTAAGTTTTGTATTTTGTTGAGACTCTAAACAAAACTGACCCGCAAGAAAGAAAAACAAACTGACAGGCAACTAACTGGTGGAACTTCCAAATCTAGTTTTTGCTGGTCATTTTTTGGTTGTTGTTGTTGTTCTGCGCATAAATATTTTAGGACGCGTATGGGAATTTTGCTCCGGGACCGTTTGTAATAGCCAAAGACTGAACTTCAACTCTCAAAGCGAGGCTCTGTTGGGCATTTGACTTTGA

基因组数据库 |小鼠登记号

NCBI登记号

运输

ambient

储存温度

−20°C

基因信息

一般描述

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

法律信息

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

储存分类代码

10 - Combustible liquids

闪点(°F)

Not applicable

闪点(°C)

Not applicable


历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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Ken-ichi Takayama et al.
Oncotarget, 6(4), 2263-2276 (2014-12-30)
Androgen receptor (AR) signaling is essential for the development of prostate cancer. Here, we report that runt-related transcription factor (RUNX1) could be a key molecule for the androgen-dependence of prostate cancer. We found RUNX1 is a target of AR and
Gillian Browne et al.
Journal of cellular physiology, 230(10), 2522-2532 (2015-03-25)
Runx1 is a transcription factor essential for definitive hematopoiesis, and genetic abnormalities in Runx1 cause leukemia. Runx1 is functionally promiscuous and acts as either an oncogene or tumor suppressor gene in certain epithelial cancers. Recent evidence suggests that Runx1 is
A de Laurentiis et al.
Oncogene, 34(49), 6018-6028 (2015-04-22)
The t(12;21) translocation is the most common genetic rearrangement in childhood acute lymphoblastic leukemia (ALL) and gives rise to the TEL-AML1 fusion gene. Many studies on TEL-AML1 describe specific properties of the fusion protein, but a thorough understanding of its

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