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About This Item
经验公式（希尔记法）:
C4H10N2O2 · HCl
CAS号:
分子量:
154.60
MDL號碼:
分類程式碼代碼:
12352209
PubChem物質ID:
NACRES:
NA.32
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品質等級
化驗
≥97% (NMR)
形狀
powder
光學活性
[α]/D +21 to +31°, c = 0.5 M in 0.1 M HCl(lit.)
儲存條件
desiccated
顏色
white to beige
溶解度
H2O: soluble (solutions may be stored for several days at 4 °C)
儲存溫度
−20°C
SMILES 字串
Cl.CNC[C@H](N)C(O)=O
InChI
1S/C4H10N2O2.ClH/c1-6-2-3(5)4(7)8;/h3,6H,2,5H2,1H3,(H,7,8);1H/t3-;/m0./s1
InChI 密鑰
VDXYGASOGLSIDM-DFWYDOINSA-N
一般說明
L-BMAA 盐酸盐/ β-N-甲基氨基-1-丙氨酸（BMAA）是一种蓝藻神经毒素（cyanobacterial neurotoxin）。
應用
L-BMAA 盐酸盐可用作标准品，用于比较沙漠结皮（desert crust）材料的β-N-甲基氨基-1-丙氨酸（BMAA）异构体分析中的所有样品。它也已被用作f/2+Sii培养基的添加剂，用于处理三角藻（Phaeodyylum tricornutum）和威氏海链藻 （Thalassiosira weissflogii）细胞，以研究其对它们的作用。
生化／生理作用
L-BMAA盐酸盐有助于阻止硫酸乙酰肝素加入磷脂酰肌醇蛋白聚糖。它具有不同寻常的谷氨酸受体结合特性。它可引起肌萎缩性侧索硬化症/ Lou Gehrig′病。
儲存類別代碼
11 - Combustible Solids
水污染物質分類（WGK）
WGK 3
閃點（°F）
Not applicable
閃點（°C）
Not applicable
個人防護裝備
Eyeshields, Gloves, type N95 (US)
The cyanobacterial neurotoxin beta-N-methylamino-l-alanine prevents addition of heparan sulfate to glypican-1 and increases processing of amyloid precursor protein in dividing neuronal cells
Cheng F, et al.
Experimental Cell Research (2019)
Boyin Yan et al.
Chemosphere, 243, 125355-125355 (2019-11-24)
Cyanobacteria produce a series of secondary metabolites, one of which is beta-N-methylamino-l-alanine (BMAA). BMAA is considered to be the cause of human neurodegeneration. Compared with other cyanotoxins, the role of BMAA in cyanobacteria remains unclear. To investigate this question, six
2-Amino-β-methylamino-propionc acid, a new amino acid from seeds of Cycas circinali.
Vega, et al.
Psychochemistry, 6, 759-762 (1967)
The effect of exogenous beta-N-methylamino-l-alanine (BMAA) on the diatoms Phaeodactylum tricornutum and Thalassiosira weissflogii
Lage S, et al.
Harmful Algae, 85-92 (2016)
Alexandra Lepoutre et al.
Toxins, 12(2) (2020-01-25)
The environmental neurotoxin β-methylamino-l-alanine (BMAA) may represent a risk for human health. BMAA accumulates in freshwater and marine organisms consumed by humans. However, few data are available about the kinetics of BMAA accumulation and detoxification in exposed organisms, as well
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