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Merck

75617

Sigma-Aldrich

2-Deoxy-L-ribose

≥97.0% (TLC)

别名:

2-Deoxy-L-erythro-pentose

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About This Item

经验公式(希尔记法):
C5H10O4
CAS号:
分子量:
134.13
MDL號碼:
分類程式碼代碼:
12352201
PubChem物質ID:
NACRES:
NA.25

化驗

≥97.0% (TLC)

形狀

powder

光學活性

[α]/D 54.0±2.0°, 24 hr, c = 1 in H2O

儲存溫度

2-8°C

SMILES 字串

O=CC[C@@H](O)[C@@H](O)CO

InChI

1S/C5H10O4/c6-2-1-4(8)5(9)3-7/h2,4-5,7-9H,1,3H2/t4-,5+/m1/s1

InChI 密鑰

ASJSAQIRZKANQN-UHNVWZDZSA-N

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生化/生理作用

Inhibitor of enzymes and valuable building block for synthesis

其他說明

To gain a comprehensive understanding of our extensive range of Monosaccharides for your research, we encourage you to visit our Carbohydrates Category page.

儲存類別代碼

11 - Combustible Solids

水污染物質分類(WGK)

WGK 3

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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Masao Shiozaki et al.
Carbohydrate research, 370, 46-66 (2013-03-05)
KRN7000 is one of the α-galactosylceramides, which has a 2-hexacosanoylamino-3,4-dihydroxyoctadecyl group. This compound, known as a ligand for the activation of CD1d mediated invariant natural killer T cells (iNKT cells) which release both T helper 1 (Th1) cytokines such as
Ryuji Ikeda et al.
Biochemical and biophysical research communications, 291(4), 806-812 (2002-02-28)
An angiogenic factor, platelet-derived endothelial cell growth factor/thymidine phosphorylase (PD-ECGF/TP), stimulates the chemotaxis of endothelial cells and confers resistance to apoptosis induced by hypoxia. 2-deoxy-D-ribose, a degradation product of thymidine generated by TP enzymatic activity partially prevented hypoxia-induced apoptosis. 2-Deoxy-D-ribose
Warfarin glycosylation invokes a switch from anticoagulant to anticancer activity.
Pauline Peltier-Pain et al.
ChemMedChem, 6(8), 1347-1350 (2011-06-30)
Aqeel Ahmed et al.
Journal of the American Chemical Society, 128(44), 14224-14225 (2006-11-02)
The reaction of 70 unprotected, diversely functionalized free reducing sugars with methoxyamine-appended colchicine led to the production of a 58-member glycorandomized library. High-throughput cytotoxicity assays revealed glycosylation to modulate specificity and potency. Library members were also identified which, unlike the
Serkan Dikici et al.
Microvascular research, 131, 104035-104035 (2020-07-01)
Delayed neovascularisation of tissue-engineered (TE) complex constructs is a major challenge that causes their failure post-implantation. Although significant progress has been made in the field of angiogenesis, ensuring rapid neovascularisation still remains a challenge. The use of pro-angiogenic agents is

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