生物来源
mouse
抗体形式
purified immunoglobulin
抗体产品类型
primary antibodies
克隆
2C6.2, monoclonal
种属反应性
rat
制造商/商品名称
Chemicon®
技术
immunohistochemistry: suitable
western blot: suitable
同位素/亚型
IgG2b
UniProt登记号
运输
wet ice
靶向翻译后修饰
unmodified
基因信息
rat ... Grin1(24408)
特异性
NMDAR1, C-terminal, C2 cassette
免疫原
Epitope: C-terminus, C2 cassette
Synthetic peptide (LQNQKDTVLPRRAIEREEGQLQLCSRHRES) corresponding to the C-terminus of rat NMDA receptor subunit.
应用
Anti-NMDAR1 Antibody, C-terminus, C2 cassette, clone 2C6.2 is an antibody against NMDAR1 for use in IH & WB.
Research Category
Neuroscience
Neuroscience
Research Sub Category
Neurotransmitters & Receptors
Neurotransmitters & Receptors
Western blot: 1:100-1:500
Immunohistochemistry: 1:100-1:500 using paraformaldehyde or paraformaldehyde/glutaraldehyde fixed tissue with light and electron microscopy.
Optimal working dilutions must be determined by the end user.
Immunohistochemistry: 1:100-1:500 using paraformaldehyde or paraformaldehyde/glutaraldehyde fixed tissue with light and electron microscopy.
Optimal working dilutions must be determined by the end user.
外形
Format: Purified
Purified immunoglobulin. Liquid in 0.02M Phosphate buffer, 0.25 M NaCl with 0.1% sodium azide.
储存及稳定性
Maintain at 2-8°C in undiluted aliquots for up to 6 months.
其他说明
Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.
法律信息
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
免责声明
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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储存分类代码
10 - Combustible liquids
WGK
WGK 2
闪点(°F)
Not applicable
闪点(°C)
Not applicable
Tom J Phillips et al.
Scientific reports, 7(1), 9079-9079 (2017-08-24)
Some neuropsychiatric disease, including schizophrenia, may originate during prenatal development, following periods of gestational hypoxia and placental oxidative stress. Here we investigated if gestational hypoxia promotes damaging secretions from the placenta that affect fetal development and whether a mitochondria-targeted antioxidant
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