推荐产品
一般說明
试剂盒包含在中性 pH 下单次反应即可从糖蛋白中去除所有 N-连接，所有简单 O-连接以及几乎所有复杂的 O-连接寡糖所需的所有酶和试剂。适用于天然或变性实验方案，不会降解蛋白质。该试剂盒已组装成包含测序级酶（不含叠氮化物、BSA 或甘油）。每个试剂盒可用于将 2 mg 糖蛋白去糖基化。
成分
牛胎球蛋白对照，变性溶液，内-α-N-乙酰半乳糖苷酶，β1,4-半乳糖苷酶，葡萄糖苷酶，N-糖苷酶 F，神经氨酸酶，反应缓冲液，TRITON™ X-100 洗涤剂和说明书。
警告
毒性：多个毒性值，请参阅 MSDS（O）
規格
试验时间：3 h-5 days
其他說明
Chiba, A., et al. 1997.J. Biol. Chem.272, 2156.
Jiang, M.S., and Hart, G.W.1997.J. Biol. Chem.272, 2421.
Wilkins, P.P., et al. 1996.J. Biol. Chem.271, 18732.
Altmann, F., et al. 1995.Glycoconj.J.12, 84.
Szkudlinski, M.W., et al. 1995.
Endocrinology136, 3325.
Pahlsson, P., et al. 1994.Glycoconj.J.11, 43.
Saito, S., et al. 1994.J. Biol. Chem.269, 5644.
Tarentino, A.L., and Plummer, T.H.1994.Methods Enzymol.230, 44.
Iwase, H., Hotta, K. 1993.Methods.Mol.Biol.14, 151.
Stults, N.L., and Cummings, R.D.1993.Glycobiology3, 589.
Hard, K., et al. 1992.Eur. J. Biochem.205, 785.
Kuraya, N., and Hase, S. 1992.J. Biochem.(Tokyo)112, 122.
Trimble, R.B., and Tarentino, A.L.1991.J. Biol. Chem.266, 1646.
Fukuda, M., et al. 1987.J. Biol. Chem.262, 11952.
Sojar, H.T., and Bahl, O.P.1987.Arch.Biochem.Biophys.259, 52.
Taga, E.M., et al. 1984.Biochemistry23, 815.
Kobata, A. 1979.Anal.Biochem.100, 1.
Uchida, Y., et al. 1979.J. Biochem (Tokyo)86, 1573.
Glasgow, L.R., et al. 1977.J. Biol. Chem.252, 8615.
Spiro, R.G., and Bhoyroo, V.D.1974.J. Biol. Chem.249, 5704.
Jiang, M.S., and Hart, G.W.1997.J. Biol. Chem.272, 2421.
Wilkins, P.P., et al. 1996.J. Biol. Chem.271, 18732.
Altmann, F., et al. 1995.Glycoconj.J.12, 84.
Szkudlinski, M.W., et al. 1995.
Endocrinology136, 3325.
Pahlsson, P., et al. 1994.Glycoconj.J.11, 43.
Saito, S., et al. 1994.J. Biol. Chem.269, 5644.
Tarentino, A.L., and Plummer, T.H.1994.Methods Enzymol.230, 44.
Iwase, H., Hotta, K. 1993.Methods.Mol.Biol.14, 151.
Stults, N.L., and Cummings, R.D.1993.Glycobiology3, 589.
Hard, K., et al. 1992.Eur. J. Biochem.205, 785.
Kuraya, N., and Hase, S. 1992.J. Biochem.(Tokyo)112, 122.
Trimble, R.B., and Tarentino, A.L.1991.J. Biol. Chem.266, 1646.
Fukuda, M., et al. 1987.J. Biol. Chem.262, 11952.
Sojar, H.T., and Bahl, O.P.1987.Arch.Biochem.Biophys.259, 52.
Taga, E.M., et al. 1984.Biochemistry23, 815.
Kobata, A. 1979.Anal.Biochem.100, 1.
Uchida, Y., et al. 1979.J. Biochem (Tokyo)86, 1573.
Glasgow, L.R., et al. 1977.J. Biol. Chem.252, 8615.
Spiro, R.G., and Bhoyroo, V.D.1974.J. Biol. Chem.249, 5704.
由于该运输中有害物质的性质，您的订单可能需要支付额外的运输费用。某些尺寸的产品可免除其他有害材料的运输费用。请与您当地的销售办事处联系，以获取有关这些费用的更多信息。
法律資訊
CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany
Triton is a trademark of The Dow Chemical Company or an affiliated company of Dow
訊號詞
Danger
危險分類
Acute Tox. 3 Dermal - Aquatic Acute 1 - Aquatic Chronic 2 - Eye Dam. 1 - Repr. 2 - Skin Irrit. 2 - Skin Sens. 1
儲存類別代碼
6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects
A Merkx-Jacques et al.
Journal of bacteriology, 186(8), 2253-2265 (2004-04-03)
flaA1 and wbpB are conserved genes with unknown biological function in Helicobacter pylori. Since both genes are predicted to be involved in lipopolysaccharide (LPS) biosynthesis, flagellum assembly, or protein glycosylation, they could play an important role in the pathogenesis of
Christoph Mück et al.
The journals of gerontology. Series A, Biological sciences and medical sciences, 65(11), 1165-1180 (2010-08-03)
Tumor necrosis factor-like cytokine 1A (TL1A) is expressed in endothelial cells and contributes to T-cell activation, via an extracellular fragment TL1A(L72-L251), generated by ectodomain shedding. Fragments of TL1A, referred to as vascular endothelial growth inhibitor, were found to induce growth
Laura E Brown et al.
The Journal of biological chemistry, 291(27), 13926-13942 (2016-04-30)
The establishment of cell-cell contacts between presynaptic GABAergic neurons and their postsynaptic targets initiates the process of GABAergic synapse formation. GABAA receptors (GABAARs), the main postsynaptic receptors for GABA, have been recently demonstrated to act as synaptogenic proteins that can
Jason A Wall et al.
American journal of physiology. Heart and circulatory physiology, 291(5), H2462-H2472 (2006-06-13)
Ischemia-reperfusion (I/R) has critical consequences in the heart. Recent studies on the functions of I/R-activated kinases, such as p38 mitogen-activated protein kinase (MAPK), showed that I/R injury is reduced in the hearts of transgenic mice that overexpress the p38 MAPK
Xiaoke Yin 殷晓科 et al.
Arteriosclerosis, thrombosis, and vascular biology, 39(9), 1859-1873 (2019-07-19)
Marfan syndrome (MFS) is caused by mutations in FBN1 (fibrillin-1), an extracellular matrix (ECM) component, which is modified post-translationally by glycosylation. This study aimed to characterize the glycoproteome of the aortic ECM from patients with MFS and relate it to
我们的科学家团队拥有各种研究领域经验，包括生命科学、材料科学、化学合成、色谱、分析及许多其他领域.
联系技术服务部门