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Merck
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16-226

Sigma-Aldrich

Anti-Nitrotyrosine Antibody, clone 1A6, Alexa Fluor 488 conjugate

clone 1A6, Upstate®, from mouse

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About This Item

分類程式碼代碼:
12352203
eCl@ss:
32160702
NACRES:
NA.41

生物源

mouse

共軛

ALEXA FLUOR 488

抗體表格

purified antibody

抗體產品種類

primary antibodies

無性繁殖

1A6, monoclonal

物種活性

vertebrates

製造商/商標名

Upstate®

技術

flow cytometry: suitable
western blot: suitable

同型

IgG2bκ

運輸包裝

wet ice

目標翻譯後修改

unmodified

基因資訊

human ... NOS1(4842)

應用

This Anti-Nitrotyrosine Antibody, clone 1A6, Alexa Fluor 488 conjugate is validated for use in FC, WB for the detection of Nitrotyrosine.

品質

Routinely evaluated by immunoblot.

外觀

Protein G Purified mouse monoclonal in a solution of 1% BSA, PBS, 0.05% Tween, and 0.05% sodium azide as a preservative.

儲存和穩定性

Stable for 1 year at 2–8°C from date of shipment in undiluted aliquots. Upon receipt, and prior to removing the cap, centrifuge the vial to remove any material in the cap. DO NOT FREEZE. Store at 2–8°C in aliquots, in the dark. Avoid repeated warming/cooling cycles.

其他說明

Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.

法律資訊

ALEXA FLUOR is a trademark of Life Technologies
UPSTATE is a registered trademark of Merck KGaA, Darmstadt, Germany

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儲存類別代碼

12 - Non Combustible Liquids

水污染物質分類(WGK)

WGK 2

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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Biological selectivity and functional aspects of protein tyrosine nitration.
Ischiropoulos, Harry
Biochemical and biophysical research communications, 305, 776-783 (2003)
Compartmentalized nitrosation and nitration in mitochondria.
Ghafourifar, Pedram and Colton, Carol A
Antioxidants & Redox Signaling, 5, 349-354 (2003)
Relevance of protein nitration in brain injury: a key pathophysiological mechanism in neurodegenerative, autoimmune, or inflammatory CNS diseases and stroke.
Sarchielli, P, et al.
Amino Acids, 25, 427-436 (2003)
J Torreilles et al.
BMC immunology, 2, 1-1 (2001-03-07)
Peroxynitrite is increasingly proposed as a contributor to defence system in marine bivalve. It can be formed by combination of superoxide and nitric oxide, and can react with tyrosine residues of proteins giving rise to 3-nitrotyrosine. The present article describes
C Ayata et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience, 17(18), 6908-6917 (1997-09-15)
We investigated the role of neuronal (type I) nitric oxide synthase (nNOS) in NMDA-mediated excitotoxicity in wild-type (SV129 and C57BL/6J) and type I NOS knock-out (nNOS-/-) mice and examined its relationship to apoptosis. Excitotoxic lesions were produced by intrastriatal stereotactic

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