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Merck

GF38388123

foil, 6mm disks, thickness 0.015mm, 99.99+%

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About This Item

线性分子式:
Fe
CAS号:
分子量:
55.85
MDL號碼:
分類程式碼代碼:
12141721
PubChem物質ID:
NACRES:
NA.23

化驗

99.99%

形狀

foil

製造商/商標名

Goodfellow 383-881-23

電阻係數

9.71 μΩ-cm

直徑× 厚度

6 mm × 0.015 mm

bp

2750 °C (lit.)

mp

1535 °C (lit.)

密度

7.86 g/mL at 25 °C (lit.)

SMILES 字串

[Fe]

InChI

1S/Fe

InChI 密鑰

XEEYBQQBJWHFJM-UHFFFAOYSA-N

一般說明

For updated SDS information please visit www.goodfellow.com.

法律資訊

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Jun-Won Jang et al.
Water science and technology : a journal of the International Association on Water Pollution Research, 59(12), 2503-2507 (2009-06-23)
Zero valent iron has been successfully used for the degradation of a wide range of contaminants. However, this reaction of using ZVI particle produces a large quantity of iron sludge. To solve the problem, we report the synthesis of self-organized
Arnold L Demain et al.
Applied microbiology and biotechnology, 73(1), 55-59 (2006-04-20)
When tetanus toxin is made by fermentation with Clostridium tetani, the traditional source of iron is an insoluble preparation called reduced iron powder. This material removes oxygen from the system by forming FeO(2) (rust). When inoculated in a newly developed
50 years ago in the Journal of Pediatrics: Iron metabolism in premature infants: II. Prevention of iron deficiency.
Pamela J Kling
The Journal of pediatrics, 164(4), 768-768 (2014-03-22)
Thomas A Russo et al.
Infection and immunity, 82(6), 2356-2367 (2014-03-26)
Hypervirulent (hypermucoviscous) Klebsiella pneumoniae (hvKP) strains are an emerging variant of "classical" K. pneumoniae (cKP) that cause organ and life-threatening infection in healthy individuals. An understanding of hvKP-specific virulence mechanisms that enabled evolution from cKP is limited. Observations by our
Eunna Choi et al.
Infection and immunity, 82(6), 2203-2210 (2014-03-20)
In order to survive inside macrophages, Salmonella produces a series of proteins encoded by genes within Salmonella pathogenicity island 2 (SPI-2). In the present study, we report that Fur, a central regulator of iron utilization, negatively controls the expression of

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