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Merck

BR701330

BRAND® 96 孔微孔板,U 形底

round bottom, non-sterile

别名:

微量板, 板子, 样品储存

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About This Item

分類程式碼代碼:
41121800
NACRES:
NB.15

材料

polypropylene
round bottom

無菌

non-sterile

包裝

pack of 100 ea

製造商/商標名

BRAND 701330

尺寸

96 well

孔最大容積

300 μm

結合類型

non-treated surface

一般說明

PP. For volumes up to 300 µl. Compatible with virtually all leading microplate centrifuges. Raised rings around the orifice of each well minimize possible cross-contamination. The plates can be sealed using self-adhesive films, such as DMSO-resistant sealing film (cross-cut) with alphanumeric coding.

特點和優勢

  • BRAND® 96 孔微孔板,U 形底是一种深孔板,非常适合凝集和其他需要搅拌和洗涤样品的测定。
  • 具有圆形井底,没有边缘。
  • 孔口周围的凸起环可防止可能的交叉污染。
  • 与一系列微孔板离心机兼容。
  • 可使用合适的密封垫或自粘密封膜牢固密封。
  • 用于准确识别的字母数字代码。
  • 由高透明度的医用级聚丙烯(PP) 制成,有助于提高样品的可见性。
  • 耐大多数溶剂,包括 DMSO、苯酚和氯仿。

法律資訊

BRAND is a registered trademark of BRAND GMBH + CO KG

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Stephen D Carter et al.
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The endoplasmic reticulum (ER) is a highly dynamic network of membranes. Here, we combine live-cell microscopy with in situ cryo-electron tomography to directly visualize ER dynamics in several secretory cell types including pancreatic β-cells and neurons under near-native conditions. Using
Gefei Chen et al.
Communications biology, 3(1), 32-32 (2020-01-22)
Molecular chaperones play important roles in preventing protein misfolding and its potentially harmful consequences. Deterioration of molecular chaperone systems upon ageing are thought to underlie age-related neurodegenerative diseases, and augmenting their activities could have therapeutic potential. The dementia relevant domain
SeongJun Han et al.
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Regulatory T cells are integral to the regulation of autoimmune and anti-tumor immune responses. However, several studies have suggested that changes in T cell signaling networks can result in T cells that are resistant to the suppressive effects of regulatory
Kirsi Savijoki et al.
Microorganisms, 8(1) (2020-01-17)
The present study investigated Staphylococcus aureus ATCC25923 surfaceomes (cell surface proteins) during prolonged growth by subjecting planktonic and biofilm cultures (initiated from exponential or stationary cells) to label-free quantitative surfaceomics and phenotypic confirmations. The abundance of adhesion, autolytic, hemolytic, and
Rosalie Heilig et al.
Life science alliance, 3(6) (2020-04-30)
Caspase-1 drives a lytic inflammatory cell death named pyroptosis by cleaving the pore-forming cell death executor gasdermin-D (GSDMD). Gsdmd deficiency, however, only delays cell lysis, indicating that caspase-1 controls alternative cell death pathways. Here, we show that in the absence

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