等級
HPLC grade
蒸汽密度
2.49 (vs air)
蒸汽壓力
71 mmHg ( 20 °C)
化驗
≥99.5%
自燃溫度
960 °F
純化經由
glass distillation
expl. lim.
10.1 %
技術
HPLC: suitable
雜質
<0.050% water
蒸發殘留物
<0.0005%
折射率
n20/D 1.379 (lit.)
bp
80 °C (lit.)
80 °C
mp
−87 °C (lit.)
密度
0.805 g/mL at 25 °C (lit.)
&lambda ;
H2O reference
紫外吸收
λ: 330 nm Amax: 1.00
λ: 340 nm Amax: 0.10
λ: 350-400 nm Amax: 0.01
SMILES 字串
CC(CC)=O
InChI
1S/C4H8O/c1-3-4(2)5/h3H2,1-2H3
InChI 密鑰
ZWEHNKRNPOVVGH-UHFFFAOYSA-N
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訊號詞
Danger
危險聲明
危險分類
Eye Irrit. 2 - Flam. Liq. 2 - STOT SE 3
標靶器官
Central nervous system
安全危害
儲存類別代碼
3 - Flammable liquids
水污染物質分類(WGK)
WGK 1
閃點(°F)
30.2 °F - closed cup
閃點(°C)
-1 °C - closed cup
個人防護裝備
Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter
Cancer research, 53(4), 721-724 (1993-02-15)
Metabolites of the tobacco-specific nitrosamine, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone, a potent pulmonary carcinogen, have been quantified in the urine of 11 smokers. They were not detected in nonsmokers' urine. The metabolites, 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanol and its glucuronide, were detected in quantities of 0.23-1.0 and 0.57-6.5
Contact dermatitis, 19(5), 348-350 (1988-11-01)
Irritant contact dermatitis along with an increased transepidermal water loss can result from exposing the skin to solvents. A study of the interaction of various solvents with human stratum corneum was made using thin-layer chromatography. Comparison of 10 solvents (trichloroethylene
Cancer immunology research, 3(6), 602-609 (2015-03-22)
Oncogene activation in tumor cells induces broad and complex cellular changes that contribute significantly to disease initiation and progression. In melanoma, oncogenic BRAF(V600E) has been shown to drive the transcription of a specific gene signature that can promote multiple mechanisms
Nature communications, 6, 7600-7600 (2015-07-22)
Transforming growth factor-β (TGF-β) and interleukin-6 (IL-6) are the pivotal cytokines to induce IL-17-producing CD4(+) T helper cells (TH17); yet their signalling network remains largely unknown. Here we show that the highly homologous TGF-β receptor-regulated Smads (R-Smads): Smad2 and Smad3
Leukemia, 29(1), 76-85 (2014-05-13)
Kinase inhibitors block proliferative signals in BCR-ABL1+ leukemic cells, but their capacity to induce apoptosis is poorly understood. Initial studies suggested that very brief exposure to kinase inhibitors was sufficient to induce apoptosis in chronic myeloid leukemia (CML) cells. However
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