推荐产品
方案
98%
折射率
n20/D 1.46 (lit.)
沸点
148-149 °C/19 mmHg (lit.)
mp
2-5 °C (lit.)
密度
0.961 g/mL at 25 °C (lit.)
SMILES字符串
CCCCCC#CC(O)=O
警示用语：
Danger
危险分类
Acute Tox. 4 Oral - Skin Corr. 1B
储存分类代码
8A - Combustible corrosive hazardous materials
WGK
WGK 3
闪点(°F)
213.8 °F - closed cup
闪点(°C)
101 °C - closed cup
个人防护装备
Faceshields, Gloves, Goggles, type ABEK (EN14387) respirator filter
Phornnop Naiyanetr et al.
Journal of autoimmunity, 37(3), 209-216 (2011-07-19)
Our laboratory has hypothesized that xenobiotic modification of the native lipoyl moiety of the major mitochondrial autoantigen, the E2 subunit of the pyruvate dehydrogenase complex (PDC-E2), may lead to loss of self-tolerance in primary biliary cirrhosis (PBC). This thesis is
Si-Jie Wu et al.
Hepatology (Baltimore, Md.), 53(3), 915-925 (2011-03-05)
Murine models of autoimmunity allow the study of the earliest events in disease pathogenesis. Our laboratory has developed a xenobiotic induced model of primary biliary cirrhosis (PBC) following immunization of mice with 2-octynoic acid coupled to bovine serum albumin (2-OA-BSA)
J M Fletcher et al.
Metabolism: clinical and experimental, 48(6), 685-688 (1999-06-25)
2-Octynoic acid was administered by intraperitoneal injection to fasted Sprague-Dawley rats in an attempt to simulate medium-chain acyl-coenzyme A dehydrogenase (MCAD) deficiency. The resultant urine organic acid profile showed a mild dicarboxylic aciduria but lacked the glycine conjugates characteristic of
J A Montgomery et al.
Biomedical & environmental mass spectrometry, 18(6), 416-423 (1989-06-01)
Rats given 2-octynoic acid by intraperitoneal injection excrete elevated amounts of medium-chain dicarboxylic acids and other acidic metabolites usually associated with human medium-chain acyl-CoA dehydrogenase deficiency. Onset of this organic acid profile is immediate and lasts for approximately 24 h.
Amy Dhirapong et al.
Hepatology (Baltimore, Md.), 57(2), 708-715 (2012-09-22)
Collectively, the data in both humans and murine models of human primary biliary cirrhosis (PBC) suggest that activated T cells, particularly CD8 T cells, play a critical role in biliary cell destruction. Under physiological conditions, T-cell activation involves two critical
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