07-210
Anti-AKAP 150 Antibody
serum, Upstate®
Synonym(s):
Anti-Anti-AKAP75, Anti-Anti-AKAP79, Anti-Anti-H21
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About This Item
UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41
Recommended Products
biological source
rabbit
Quality Level
antibody form
serum
antibody product type
primary antibodies
clone
polyclonal
species reactivity
rat
manufacturer/tradename
Upstate®
technique(s)
western blot: suitable
isotype
IgG
NCBI accession no.
UniProt accession no.
shipped in
dry ice
target post-translational modification
unmodified
Gene Information
human ... AKAP1(8165)
Specificity
AKAP 150
Immunogen
peptide corresponding to amino acids 428-449 of rat AKAP 150 (C-TTVGQAEEATVGQAEEATVGQA)
Application
Anti-AKAP 150 Antibody is an antibody against AKAP 150 for use in WB.
Research Category
Signaling
Signaling
Research Sub Category
Kinases & Phosphatases
RNA Binding Protein (RBP)
Kinases & Phosphatases
RNA Binding Protein (RBP)
Quality
routinely evaluated by immunoblot on a rat brain cytosolic preparation
Target description
150kDa
Physical form
0.1M Tris-glycine, pH 7.4, 0.15M NaCl, 0.05% sodium azide before the addition of glycerol to 30%
Antiserum
Storage and Stability
2 years at -20°C
Legal Information
UPSTATE is a registered trademark of Merck KGaA, Darmstadt, Germany
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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Storage Class Code
10 - Combustible liquids
WGK
WGK 1
Certificates of Analysis (COA)
Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.
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AKAP signaling in reinstated cocaine seeking revealed by iTRAQ proteomic analysis.
Reissner, KJ; Uys, JD; Schwacke, JH; Comte-Walters, S; Rutherford-Bethard, JL; Dunn et al.
The Journal of Neuroscience null
Matthew A Nystoriak et al.
Science signaling, 10(463) (2017-01-26)
Hypercontractility of arterial myocytes and enhanced vascular tone during diabetes are, in part, attributed to the effects of increased glucose (hyperglycemia) on L-type CaV1.2 channels. In murine arterial myocytes, kinase-dependent mechanisms mediate the increase in CaV1.2 activity in response to
Aleksandra Polishchuk et al.
Cell communication and signaling : CCS, 22(1), 371-371 (2024-07-24)
Protein kinase A (PKA) enhances neurotransmission at the neuromuscular junction (NMJ), which is retrogradely regulated by nerve-induced muscle contraction to promote Acetylcholine (ACh) release through the phosphorylation of molecules involved in synaptic vesicle exocytosis (SNAP-25 and Synapsin-1). However, the molecular
Christopher H Bohrer et al.
Nature methods, 18(6), 669-677 (2021-06-02)
Single-molecule localization microscopy (SMLM) relies on the blinking behavior of a fluorophore, which is the stochastic switching between fluorescent and dark states. Blinking creates multiple localizations belonging to the same fluorophore, confounding quantitative analyses and interpretations. Here we present a
Bo Chen et al.
Journal of neuroinflammation, 19(1), 181-181 (2022-07-14)
Perioperative neurocognitive disorders (PNDs) are considered the most common postoperative complication in geriatric patients. However, its pathogenesis is not fully understood. Surgery-triggered neuroinflammation is a major contributor to the development of PNDs. Neuroinflammation can influence N-methyl-D-aspartate receptor (NMDAR) expression or
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