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D9753

Sigma-Aldrich

DL-3,4-Dihydroxyphenyl glycol

Synonym(s):

DL-α,β,3,4-Tetrahydroxyphenethyl alcohol, DL-β,3,4-Trihydroxyphenethyl alcohol

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About This Item

Empirical Formula (Hill Notation):
C8H10O4
CAS Number:
28822-73-3
Molecular Weight:
170.16
EC Number:
249-260-9
MDL number:
MFCD00004702
UNSPSC Code:
12352106
PubChem Substance ID:
24894324
NACRES:
NA.25

form

powder

storage temp.

−20°C

SMILES string

OCC(O)c1ccc(O)c(O)c1

InChI

1S/C8H10O4/c9-4-8(12)5-1-2-6(10)7(11)3-5/h1-3,8-12H,4H2

InChI key

MTVWFVDWRVYDOR-UHFFFAOYSA-N

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Pictograms

Exclamation mark
GHS07

Signal Word

Warning

Hazard Statements

H319

Hazard Classifications

Eye Irrit. 2

Storage Class Code

11 - Combustible Solids

WGK

WGK 3

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Certificates of Analysis (COA)

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W A Divers et al.
American journal of obstetrics and gynecology, 141(6), 625-628 (1981-11-15)
To assess the effect of maternal smoking on fetal adrenergic activity, simultaneous measurements in amniotic fluid of the parent catecholamines, dopamine (DA), norepinephrine (NE), and epinephrine (E), as well as the specific intraneuronal deaminated metabolites of DA, 3,4-dihydroxyphenylacetic acid (DOPAC)
Graeme Eisenhofer et al.
Pharmacological reviews, 56(3), 331-349 (2004-08-20)
This article provides an update about catecholamine metabolism, with emphasis on correcting common misconceptions relevant to catecholamine systems in health and disease. Importantly, most metabolism of catecholamines takes place within the same cells where the amines are synthesized. This mainly
David S Goldstein et al.
Brain : a journal of neurology, 135(Pt 6), 1900-1913 (2012-03-28)
Central catecholamine deficiency characterizes α-synucleinopathies such as Parkinson's disease. We hypothesized that cerebrospinal fluid levels of neuronal metabolites of catecholamines provide neurochemical biomarkers of these disorders. To test this hypothesis we measured cerebrospinal fluid levels of catechols including dopamine, norepinephrine
Osnat Ben-Shahar et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience, 33(2), 495-506a-495-506a (2013-01-11)
Anomalies in prefrontal cortex (PFC) function are posited to underpin difficulties in learning to suppress drug-seeking behavior during abstinence. Because group 1 metabotropic glutamate receptors (mGluRs) regulate drug-related learning, we assayed the consequences of extended access to intravenous cocaine (6
Adrianne R Wilson-Poe et al.
Neuropharmacology, 66, 348-354 (2012-07-10)
Metabotropic glutamate (mGlu) receptors modulate pain from within the midbrain periaqueductal grey (PAG). In the present study, the postsynaptic mGlu receptor mediated effects on rat PAG neurons were examined using whole-cell patch-clamp recordings in brain slices. The selective group I
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