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Merck

P6617

Sigma-Aldrich

Pristanic acid

≥97% (GC), ethanol solution

Sinónimos:

2,6,10,14-Tetramethylpentadecanoic acid

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About This Item

Fórmula empírica (notación de Hill):
C19H38O2
Número de CAS:
Peso molecular:
298.50
EC Number:
MDL number:
UNSPSC Code:
12352106
PubChem Substance ID:
NACRES:
NA.77

Nombre del producto

Pristanic acid solution, mixture of isomers, ethanol solution, ≥97% (GC)

Quality Level

assay

≥97% (GC)

form

ethanol solution

storage temp.

−20°C

SMILES string

CC(C)CCCC(C)CCCC(C)CCCC(C)C(O)=O

InChI

1S/C19H38O2/c1-15(2)9-6-10-16(3)11-7-12-17(4)13-8-14-18(5)19(20)21/h15-18H,6-14H2,1-5H3,(H,20,21)

InChI key

PAHGJZDQXIOYTH-UHFFFAOYSA-N

Biochem/physiol Actions

Oxidation product of fatty acids in the diet, particularly phytanic acid. Phytanic acid is oxidized by alpha oxidation yielding pristanic acid, which is subsequently degraded by peroxisomal beta oxidation. Several inborn errors of metabolism with one or more deficiencies in the phytanic acid and pristanic acid breakdown have been described.

pictograms

FlameExclamation mark

signalword

Danger

hcodes

Hazard Classifications

Eye Irrit. 2 - Flam. Liq. 2

Storage Class

3 - Flammable liquids

wgk_germany

WGK 2

flash_point_f

55.4 °F

flash_point_c

13 °C

ppe

Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter


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Los clientes también vieron

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The differentiation of carnitine-acylcarnitine translocase deficiency (CACT) from carnitine palmitoyltransferase type II deficiency (CPT-II) and long-chain 3-hydroxyacyl-CoA dehydrogenase (LCHAD) deficiency from mitochondrial trifunctional protein deficiency (MTP) continues to be ambiguous using current acylcarnitine profiling techniques either from plasma or blood
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R J Wanders et al.
European journal of pediatrics, 153(7 Suppl 1), S44-S48 (1994-01-01)
Peroxisomal disorders represent a recently recognized group of inherited diseases in man, now comprising 14 different disorders. If discussion is restricted to those peroxisomal disorders in which there is neurological involvement (thereby excluding hyperoxaluria and acatalasaemia), results over the least
Antonella Peduto et al.
Molecular genetics and metabolism, 82(3), 224-230 (2004-07-06)
Peroxisomal disorders include a complex spectrum of diseases, characterized by a high heterogeneity from both the clinical and the biochemical points of view. Specific assays are required for the study of peroxisome metabolism. Among these, pipecolic acid evaluation is considered

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