05-862
Anti-Plasminogen/Angiostatin Antibody, clone GMA-013
clone GMA-013, Upstate®, from mouse
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100 μG
€336.00
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100 μG
€336.00
About This Item
UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41
Recommended Products
biological source
mouse
Quality Level
antibody form
purified immunoglobulin
antibody product type
primary antibodies
clone
GMA-013, monoclonal
species reactivity
human
manufacturer/tradename
Upstate®
technique(s)
ELISA: suitable
western blot: suitable
isotype
IgM
NCBI accession no.
UniProt accession no.
shipped in
dry ice
target post-translational modification
unmodified
Gene Information
human ... PLG(5340)
Specificity
Plasminogen (kringles 1-3)
Immunogen
Purified human plasminogen
Application
Research Category
Inflammation & Immunology
Inflammation & Immunology
Research Sub Category
Inflammation & Autoimmune Mechanisms
Inflammation & Autoimmune Mechanisms
This Anti-Plasminogen/Angiostatin Antibody, clone GMA-013 is validated for use in ELISA, WB for the detection of Plasminogen/Angiostatin.
Quality
routinely evaluated by immunoblot on human plasminogen
Target description
Mr 88kDa
Physical form
DEAE purified
Format: Purified
Storage and Stability
stable 2 years at -20°C from date of shipment
Legal Information
UPSTATE is a registered trademark of Merck KGaA, Darmstadt, Germany
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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Storage Class Code
11 - Combustible Solids
WGK
WGK 3
Certificates of Analysis (COA)
Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.
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Angiostatin and anti-angiogenic therapy in human disease.
Wahl, Miriam L, et al.
Recent Progress in Hormone Research, 59, 73-104 (2004)
Christopher D Barrett et al.
The journal of trauma and acute care surgery, 83(6), 1053-1061 (2017-08-25)
A subset of trauma patients undergo fibrinolysis shutdown rather than pathologic hyperfibrinolysis, contributing to organ failure. The molecular basis for fibrinolysis shutdown in trauma is incompletely understood. Elastase released from primed/activated human neutrophils (HNE) has historically been described as fibrin(ogen)olytic.
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