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Key Documents

EHU031601

Sigma-Aldrich

MISSION® esiRNA

targeting human GPR19

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About This Item

UNSPSC Code:
41105324
NACRES:
NA.51

description

Powered by Eupheria Biotech

Quality Level

product line

MISSION®

form

lyophilized powder

esiRNA cDNA target sequence

CAAGGGCTATTCCTGACCAAGAAGATCTAAAGAGAACGTCTCTGAAATCAAGTCCGGATGAAGAATTAAGAGAAAAAAAGTGAATATGGTTTTTGCTCACAGAATGGATAACAGCAAGCCACATTTGATTATTCCTACACTTCTGGTGCCCCTCCAAAACCGCAGCTGCACTGAAACAGCCACACCTCTGCCAAGCCAATACCTGATGGAATTAAGTGAGGAGCACAGTTGGATGAGCAACCAAACAGACCTTCACTATGTGCTGAAACCCGGGGAAGTGGCCACAGCCAGCATCTTCTTTGGGATTCTGTGGTTGTTTTCTATCTTCGGCAATTCCCTGGTTTGTTTGGTCATCCATAGGAGTAGGAGGACTCAGTCTACCACCAACTACTTTGTGGTCTCCATGGCATGTGCTGACCTTCTCATCAGCGTTGCCAGCACGCCTTTCGTCCTGCTCCAGTTCACCACTGGAAGGTGG

Ensembl | human accession no.

NCBI accession no.

shipped in

ambient

storage temp.

−20°C

Gene Information

Related Categories

General description

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Legal Information

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

Storage Class

10 - Combustible liquids

flash_point_f

Not applicable

flash_point_c

Not applicable


Certificates of Analysis (COA)

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Lauren M Stein et al.
American journal of physiology. Regulatory, integrative and comparative physiology, 310(6), R476-R480 (2016-01-08)
Adropin, a recently described peptide hormone produced in the brain and liver, has been reported to have physiologically relevant actions on glucose homeostasis and lipogenesis, and to exert significant effect on endothelial function. We describe a central nervous system action
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Redox biology, 18, 25-32 (2018-06-18)
Mitochondria supply ~90% of the ATP required for contractile function in cardiac cells. While adult cardiomyocytes preferentially utilize fatty acids as a fuel source for oxidative phosphorylation, cardiac mitochondria can switch to other substrates when required. This change is driven

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