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Merck

AB9678

Anti-GABA A Receptor α 5 Antibody

Chemicon®, from rabbit

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100 μL

2530,00 zł

2530,00 zł

Przewidywany termin wysyłki25 marca 2026Szczegóły

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Informacje o tej pozycji

UNSPSC Code:
12352203
NACRES:
NA.41
eCl@ss:
32160702
Conjugate:
unconjugated
Clone:
polyclonal
Application:
WB
Citations:
17

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biological source

rabbit

Quality Level

100

conjugate

unconjugated

antibody form

affinity purified immunoglobulin

antibody product type

primary antibodies

clone

polyclonal

purified by

affinity chromatography

species reactivity

rat, mouse

manufacturer/tradename

Chemicon®

technique(s)

western blot: suitable

NCBI accession no.

NM_000810.2

UniProt accession no.

P31644

shipped in

dry ice

target post-translational modification

unmodified

Gene Information

human ... GABRA5(2558)

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Ta pozycja
SAB2100878SAB5202248SAB5202242
Anti-GABA A Receptor α 5 Antibody Chemicon®, from rabbit

Sigma-Aldrich

AB9678

Anti-GABA A Receptor α 5 Antibody

Anti-GABRA5 (ab2) antibody produced in rabbit affinity isolated antibody

Sigma-Aldrich

SAB2100878

Anti-GABRA5 (ab2) antibody produced in rabbit

Monoclonal Anti-GABA A Receptor-Allophycocyanin antibody produced in mouse clone S415-24

Sigma-Aldrich

SAB5202248

Monoclonal Anti-GABA A Receptor-Allophycocyanin antibody produced in mouse

Monoclonal Anti-GABA A Receptor-Atto 594 antibody produced in mouse clone S415-24

Sigma-Aldrich

SAB5202242

Monoclonal Anti-GABA A Receptor-Atto 594 antibody produced in mouse

Quality Level

100

Quality Level

100

Quality Level

-

Quality Level

-

antibody form

affinity purified immunoglobulin

antibody form

affinity isolated antibody

antibody form

purified immunoglobulin

antibody form

purified immunoglobulin

biological source

rabbit

biological source

rabbit

biological source

mouse

biological source

mouse

UniProt accession no.

P31644

UniProt accession no.

P31644

UniProt accession no.

-

UniProt accession no.

-

species reactivity

rat, mouse

species reactivity

mouse, guinea pig, rabbit, rat, human

species reactivity

human, mouse

species reactivity

human, mouse

Gene Information

human ... GABRA5(2558)

Gene Information

human ... GABRA5(2558)

Gene Information

human ... GABRA5(2558)

Gene Information

human ... GABRA5(2558)

Immunogen

Fusion protein from the Cytosolic loop of rat the GABAA Receptor alpha 5 subunit.

Application

Anti-GABA A Receptor α 5 Antibody is an antibody against GABA A Receptor α 5 for use in WB.

Biochem/physiol Actions

GABAA Receptor alpha 5 subunit. The antibody recognizes a protein of ~55 kDa corresponding to GABAA Receptor alpha 5 subunit in lysates from rat hippocampus and thalamus.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
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Klasa składowania

10 - Combustible liquids

wgk

WGK 2

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Dokumenty związane z niedawno zakupionymi produktami zostały zamieszczone w Bibliotece dokumentów.

Odwiedź Bibliotekę dokumentów

Liang Li et al.
Frontiers in neurology, 12, 591223-591223 (2021-03-13)
Post-operative cognitive dysfunction (POCD) is a common complication during the post-operative period. It affects the recovery time of the patient after surgery and the stay time in hospital, which causes a great deal of burden to patients and families emotionally
Kohei Koga et al.
Molecular pain, 14, 1744806918783478-1744806918783478 (2018-06-30)
Background Chronic pain is a persistent unpleasant sensation that produces pathological synaptic plasticity in the central nervous system. Both human imaging study and animal studies consistently demonstrate that the anterior cingulate cortex is a critical cortical area for nociceptive and
George Gallos et al.
American journal of physiology. Lung cellular and molecular physiology, 308(9), L931-L942 (2015-02-11)
The clinical need for novel bronchodilators for the treatment of bronchoconstrictive diseases remains a major medical issue. Modulation of airway smooth muscle (ASM) chloride via GABAA receptor activation to achieve relaxation of precontracted ASM represents a potentially beneficial therapeutic option.
Muhammad Hassan et al.
International journal of molecular sciences, 23(24) (2022-12-24)
Absence seizures are hyperexcitations within the cortico-thalamocortical (CTC) network, however the underlying causative mechanisms at the cellular and molecular level are still being elucidated and appear to be multifactorial. Dysfunctional feed-forward inhibition (FFI) is implicated as one cause of absence
Guobin Xia et al.
Molecular psychiatry, 26(7), 2837-2853 (2021-03-27)
The high comorbidity between obesity and mental disorders, such as depression and anxiety, often exacerbates metabolic and neurological symptoms significantly. However, neural mechanisms that underlie reciprocal control of feeding and mental states are largely elusive. Here we report that melanocortin
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SKUNUMER GTIN
AB967804053252511479

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