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Merck
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MAB5608

Sigma-Aldrich

Anti-GT1b Ganglioside Antibody

ascites fluid, clone GT1b-2b, Chemicon®

Sinónimos:

Anti-Ganglioside GT1b, GT1b Detection Antibody

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About This Item

UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41

biological source

mouse

Quality Level

100

antibody form

ascites fluid

antibody product type

primary antibodies

clone

GT1b-2b, monoclonal

species reactivity

rat, human, mouse

manufacturer/tradename

Chemicon®

technique(s)

immunohistochemistry: suitable

isotype

IgG2b

shipped in

dry ice

target post-translational modification

unmodified

Specificity

GT1b ganglioside. Stains myelinated motor and sensory axons in spinal roots and peripheral nerves.

Immunogen

Purified GT1b ganglioside

Application

Anti-GT1b Ganglioside Antibody is an antibody against GT1b Ganglioside for use in IH.
Research Category
Neuroscience
Research Sub Category
Neuronal & Glial Markers

Physical form

Ascites
Liquid. Contains no preservative.

Storage and Stability

Maintain at -20°C in undiluted aliquots for up to 6 months after date of receipt. Avoid repeated freeze/thaw cycles.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Storage Class

10 - Combustible liquids

wgk_germany

WGK 1

flash_point_f

Not applicable

flash_point_c

Not applicable

Certificados de análisis (COA)

Busque Certificados de análisis (COA) introduciendo el número de lote del producto. Los números de lote se encuentran en la etiqueta del producto después de las palabras «Lot» o «Batch»

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Vardit Rubovitch et al.
Scientific reports, 7, 41269-41269 (2017-01-24)
Blast induced traumatic brain injury (B-TBI) may cause various degrees of cognitive and behavioral disturbances but the exact brain pathophysiology involved is poorly understood. It was previously suggested that ganglioside alteration on the axon surface as well as axonal regenerating
Richard Desplantes et al.
Scientific reports, 7(1), 1032-1032 (2017-04-23)
The development of simple molecular assays with membrane protein receptors in a native conformation still represents a challenging task. Exosomes are extracellular vesicles which, due to their stability and small size, are suited for analysis in various assay formats. Here
Hyoungsub Lim et al.
The EMBO journal, 39(6), e102214-e102214 (2020-02-08)
Spinal cord microglia contribute to nerve injury-induced neuropathic pain. We have previously demonstrated that toll-like receptor 2 (TLR2) signaling is critical for nerve injury-induced activation of spinal cord microglia, but the responsible endogenous TLR2 agonist has not been identified. Here
Alba Di Pardo et al.
Frontiers in neuroscience, 10, 457-457 (2016-10-22)
Huntington Disease (HD) is a genetic neurodegenerative disorder characterized by broad types of cellular and molecular dysfunctions that may affect both neuronal and non-neuronal cell populations. Among all the molecular mechanisms underlying the complex pathogenesis of the disease, alteration of
Ceyda Caliskan et al.
ALTEX (2024-07-29)
Botulinum neurotoxin type A (BoNT/A) is a widely used biopharmaceutic for the treatment of neurological diseases and aesthetic medicine, allowing months-long paralysis of target muscles and glands. Large numbers of mice are used for multiple botulinum applications including batch release
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