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Sigma-Aldrich

L-Aspartic acid-13C4

98 atom % 13C, 95% (CP)

Synonym(s):

(S)-(+)-Aminosuccinic acid-13C4, (S)-Aminobutanedioic acid-13C4, 13C Labeled L-aspartic acid

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About This Item

Linear Formula:
HO213C13CH213CH(NH2)13CO2H
CAS Number:
Molecular Weight:
137.07
MDL number:
UNSPSC Code:
12352209
PubChem Substance ID:
NACRES:
NA.12

isotopic purity

98 atom % 13C

Quality Level

Assay

95% (CP)

form

solid

optical activity

[α]25/D +25.0°, c = 2 in 5 M HCl

mp

>300 °C (dec.) (lit.)

mass shift

M+4

SMILES string

N[13C@@H]([13CH2][13C](O)=O)[13C](O)=O

InChI

1S/C4H7NO4/c5-2(4(8)9)1-3(6)7/h2H,1,5H2,(H,6,7)(H,8,9)/t2-/m0/s1/i1+1,2+1,3+1,4+1

InChI key

CKLJMWTZIZZHCS-UVYXLFMMSA-N

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General description

L-Aspartic acid-13C4 is an isotope-labeled analog of aspartic acid, wherein carbon atoms are replaced by 13C.

Packaging

This product may be available from bulk stock and can be packaged on demand. For information on pricing, availability and packaging, please contact Stable Isotopes Customer Service.

Storage Class Code

11 - Combustible Solids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Maternal dietary of n-3 polyunsaturated fatty acids affects the neurogenesis and neurochemical in female rat at weaning
Tang M, et al.
advanced synthesis and catalysis, 128, 11-20 (2018)
Maternal dietary of n-3 polyunsaturated fatty acids affects the neurogenesis and neurochemical in female rat at weaning
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Numerous mechanisms to support cells under conditions of transient nutrient starvation have been described. Several functions of the tumor-suppressor protein p53 can contribute to the adaptation of cells to metabolic stress and help cancer cell survival under nutrient-limiting conditions. We
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Dysregulation of extracellular matrix (ECM) deposition and cellular metabolism promotes tumor aggressiveness by sustaining the activity of key growth, invasion, and survival pathways. Yet mechanisms by which biophysical properties of ECM relate to metabolic processes and tumor progression remain undefined.

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