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05-5895

Sigma-Aldrich

Copper(II) chloride

SAJ first grade, ≥98.0%

Synonym(s):

Cupric chloride

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About This Item

Linear Formula:
CuCl2
CAS Number:
7447-39-4
Molecular Weight:
134.45
MDL number:
MFCD00010972
UNSPSC Code:
12352302
PubChem Substance ID:
329748107

grade

SAJ first grade

Assay

≥98.0%

form

solid

reaction suitability

reagent type: catalyst
core: copper

availability

available only in Japan

mp

620 °C (lit.)

density

3.386 g/mL at 25 °C (lit.)

storage temp.

15-25°C

SMILES string

Cl[Cu]Cl

InChI

1S/2ClH.Cu/h2*1H;/q;;+2/p-2

InChI key

ORTQZVOHEJQUHG-UHFFFAOYSA-L

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Signal Word

Danger

Hazard Classifications

Acute Tox. 4 Dermal - Acute Tox. 4 Oral - Aquatic Acute 1 - Aquatic Chronic 2 - Eye Dam. 1 - Skin Irrit. 2

Storage Class Code

8A - Combustible corrosive hazardous materials

WGK

WGK 3

Certificates of Analysis (COA)

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Iron(II)-catalyzed trifluoromethylation of potassium vinyltrifluoroborates.
Andrew T Parsons et al.
Angewandte Chemie (International ed. in English), 51(12), 2947-2950 (2012-02-14)
Francesca Cima et al.
Chemosphere, 89(1), 19-29 (2012-05-01)
After the widespread ban of TBT, due to its severe impact on coastal biocoenoses, mainly related to its immunosuppressive effects on both invertebrates and vertebrates, alternative biocides such as Cu(I) salts and the triazine Irgarol 1051, the latter previously used
Pieter Spincemaille et al.
Toxicology and applied pharmacology, 280(2), 345-351 (2014-08-20)
Wilson disease (WD) is caused by accumulation of excess copper (Cu) due to a mutation in the gene encoding the liver Cu transporter ATP7B, and is characterized by acute liver failure or cirrhosis and neuronal cell death. We investigated the
Kenji Funaki et al.
Organic letters, 14(24), 6186-6189 (2012-12-12)
Direct arylation of thiophenes and benzothiophenes with aryltrimethylsilanes was effectively catalyzed by PdCl(2)(MeCN)(2) in the presence of CuCl(2) as an oxidant. The reaction preferentially occurred at the β-position of both thiophenes and benzothiophenes.
Su-Jeong Kim et al.
Free radical biology & medicine, 53(4), 936-950 (2012-06-12)
Loss-of-function mutations in the PARK7/DJ-1 gene cause early onset autosomal-recessive Parkinson disease. DJ-1 has been implicated in protection of neurons from oxidative stress and in regulation of transcriptional activity. However, whether there is a relationship between the subcellular localization of
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