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Z373508

Sigma-Aldrich

Carestream® BioMax® light film

Synonym(s):

autoradiography film, xray film

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About This Item

UNSPSC Code:
41105341
NACRES:
NB.22

size

7 in. (18 cm) × 9.5 in. (24 cm)

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General description

Sheets of film with no interleaved paper
Double-emulsion film on clear base. Provides clearest background and highest sensitivity for chemiluminescence detection systems. Also suitable for autoradiography, but with less sensitivity for high-energy isotopes than X-Omat AR and BioMax MS, and than BioMax MR for medium-energy isotopes.

Legal Information

BioMax is a registered trademark of Carestream Health, Inc.
Carestream is a registered trademark of Carestream Health, Inc.
Kodak is a registered trademark of Eastman Kodak Company

Certificates of Analysis (COA)

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Steven C Chen et al.
PloS one, 6(5), e19780-e19780 (2011-05-24)
Although recent publications have linked the molecular events driving facioscapulohumeral muscular dystrophy (FSHD) to expression of the double homeobox transcription factor DUX4, overexpression of FRG1 has been proposed as one alternative causal agent as mice overexpressing FRG1 present with muscular
Matthias Höllerhage et al.
Scientific reports, 7(1), 11469-11469 (2017-09-15)
α-synuclein-induced neurotoxicity is a core pathogenic event in neurodegenerative synucleinopathies such as Parkinson's disease, dementia with Lewy bodies, or multiple system atrophy. There is currently no disease-modifying therapy available for these diseases. We screened 1,600 FDA-approved drugs for their efficacy
Shaista Naqvi et al.
Proceedings of the National Academy of Sciences of the United States of America, 106(19), 7762-7767 (2009-05-07)
Vitamin deficiency affects up to 50% of the world's population, disproportionately impacting on developing countries where populations endure monotonous, cereal-rich diets. Transgenic plants offer an effective way to increase the vitamin content of staple crops, but thus far it has
Sudhakar Raja Subramaniam et al.
Neurobiology of disease, 70, 204-213 (2014-07-13)
Parkinson's disease (PD) is characterized by the progressive degeneration of nigrostriatal dopaminergic neurons leading to motor deficits. The mechanisms underlying the preferential vulnerability of nigrostriatal dopaminergic neurons in PD remain poorly understood. Recent evidence supports a role for mitochondrial dysfunction

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