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S0945

Anti-Somatostatin Receptor Type 4 antibody produced in rabbit

Name: Anti-Somatostatin Receptor Type 4 antibody produced in rabbit
Brand: SIGMA

affinity isolated antibody, buffered aqueous solution

Sinónimos:

Anti-SSTR4

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About This Item

Número MDL:

MFCD06798543

Código UNSPSC:

12352203

NACRES:

NA.41

origen biológico

rabbit

Nivel de calidad

200

conjugado

unconjugated

forma del anticuerpo

affinity isolated antibody

tipo de anticuerpo

primary antibodies

clon

polyclonal

Formulario

buffered aqueous solution

reactividad de especies

human

disponibilidad

not available in Japan

técnicas

immunohistochemistry (formalin-fixed, paraffin-embedded sections): 3 μg/mL using human pancreas, Islets of Langerhans

Nº de acceso UniProt

P31391

Condiciones de envío

dry ice

temp. de almacenamiento

−20°C

modificación del objetivo postraduccional

unmodified

Información sobre el gen

human ... SSTR4(6754)
mouse ... Sstr4(20608)
rat ... Sstr4(25555)

Inmunógeno

synthetic peptide corresponding to the N-terminal extracellular domain of human somatostatin receptor type 4, conjugated to KLH. The immunizing peptide has 50% homology with the rat gene and 38% homology with the mouse gene.

Forma física

Solution in phosphate buffered saline, pH 7.7, containing 0.01% sodium azide.

Cláusula de descargo de responsabilidad

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Código de clase de almacenamiento

10 - Combustible liquids

Clase de riesgo para el agua (WGK)

nwg

Punto de inflamabilidad (°F)

Not applicable

Punto de inflamabilidad (°C)

Not applicable

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Does somatostatin have a role in the regulation of cortisol secretion in primary pigmented nodular adrenocortical disease (ppnad)? a clinical and in vitro investigation.

Zakariae Bram et al.

The Journal of clinical endocrinology and metabolism, 99(5), E891-E901 (2014-02-12)

Somatostatin (SST) receptors (SSTRs) are expressed in a number of tissues, including the adrenal cortex, but their role in cortisol secretion has not been well characterized. The objective of the study was to investigate the expression of SSTRs in the

Effect of intestinal inflammation on the cell-specific expression of somatostatin receptor subtypes in the murine ileum.

J Van Op den Bosch et al.

Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society, 19(7), 596-606 (2007-06-27)

Despite our knowledge of somatostatin (SOM) in gastrointestinal functions, little information is available on the SOM receptors (SSTRs) mediating these effects. This study focussed on the expression of SSTRs in non-inflamed and Schistosoma mansoni-infected murine ileum using immunocytochemistry, reverse transcriptase

Effect of genetic SSTR4 ablation on inflammatory peptide and receptor expression in the non-inflamed and inflamed murine intestine.

Joeri Van Op den Bosch et al.

Journal of cellular and molecular medicine, 13(9B), 3283-3295 (2009-05-12)

The recently suggested pivotal role of somatostatin (SOM) receptor 4 (SSTR4) in inflammation and nociception in several non-intestinal organs and in gastrointestinal (GI) physiology, necessitates exploration of the role of SSTR4 in GI pathophysiology. Therefore, the role of SSTR4 in

Regulation of somatostatin receptor 4-mediated cytostatic effects by CD26 in malignant pleural mesothelioma.

J Yamamoto et al.

British journal of cancer, 110(9), 2232-2245 (2014-04-20)

Malignant pleural mesothelioma (MPM) is an aggressive neoplasm arising from mesothelial lining of pleura. CD26 molecules preferentially expressed on epithelioid type of MPM. This study investigates the molecular mechanisms of CD26 regulating MPM cells in vitro and in vivo. Biochemical

Somatostatin modulates mast cell-induced responses in murine spinal neurons and satellite cells.

Joeri Van Op den bosch et al.

American journal of physiology. Gastrointestinal and liver physiology, 297(2), G406-G417 (2009-05-30)

The course of intestinal inflammatory responses is tightly coordinated by the extensive communication between the immune system and the enteric nervous system, among which the bidirectional mast cell-neuron interaction within the intestinal wall plays a prominent role. Recent research suggests

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