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Merck
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GHS216

Sigma-Aldrich

Hematoxylin Solution, Gill No. 2

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About This Item

MDL number:
MFCD00283323
UNSPSC Code:
41116124
NACRES:
NA.47

form

solution

Quality Level

500

shelf life

Expiry date on the label.

IVD

for in vitro diagnostic use

concentration

4 g/L

pH

2.5-3.5

application(s)

hematology
histology

storage temp.

room temp

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Application

Gill formulation No. 2 may be used for cytology and/or histology as a progressive or regressive stain depending on length of staining time.

Other Notes

4 g/L certified hematoxylin

pictograms

Corrosion
GHS05

signalword

Danger

hcodes

H318

Hazard Classifications

Eye Dam. 1

Storage Class

12 - Non Combustible Liquids

wgk_germany

WGK 1

flash_point_f

Not applicable

flash_point_c

Not applicable

Certificados de análisis (COA)

Busque Certificados de análisis (COA) introduciendo el número de lote del producto. Los números de lote se encuentran en la etiqueta del producto después de las palabras «Lot» o «Batch»

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Haley R Noonan et al.
Disease models & mechanisms, 9(8), 873-884 (2016-08-05)
Patients with von Hippel-Lindau (VHL) disease harbor a germline mutation in the VHL gene leading to the development of several tumor types including clear cell renal cell carcinoma (ccRCC). In addition, the VHL gene is inactivated in over 90% of
Yitzhak Reizel et al.
Genes & development, 34(15-16), 1039-1050 (2020-06-21)
The FoxA transcription factors are critical for liver development through their pioneering activity, which initiates a highly complex regulatory network thought to become progressively resistant to the loss of any individual hepatic transcription factor via mutual redundancy. To investigate the
G Gonzalez et al.
Diseases of the esophagus : official journal of the International Society for Diseases of the Esophagus, 29(6), 670-680 (2015-08-08)
Deep esophageal glands play a vital role in the protection and regeneration of the esophageal mucosa. Conditions such as gastroesophageal reflux disease and Barrett's esophagus have been associated with a change in the usual glands by oncocytic metaplasia. However, little
Wajiha Gohir et al.
The Journal of physiology, 597(12), 3029-3051 (2019-05-14)
Maternal obesity has been associated with shifts in intestinal microbiota, which may contribute to impaired barrier function Impaired barrier function may expose the placenta and fetus to pro-inflammatory mediators We investigated the impacts of diet-induced obesity in mice on maternal
Noora Puhakka et al.
PloS one, 12(3), e0172521-e0172521 (2017-03-09)
Traumatic brain injury (TBI) can result in several dentate gyrus-regulated disabilities. Almost nothing is known about the chronic molecular changes after TBI, and their potential as treatment targets. We hypothesized that chronic transcriptional alterations after TBI are under microRNA (miRNA)
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