A3172
2,5-Anhydro-D-mannitol
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About This Item
Fórmula empírica (notación de Hill):
C6H12O5
Número de CAS:
Peso molecular:
164.16
EC Number:
MDL number:
UNSPSC Code:
12352201
PubChem Substance ID:
mp
101-103 °C (lit.)
storage temp.
2-8°C
SMILES string
OCC1OC(CO)C(O)C1O
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Storage Class
13 - Non Combustible Solids
wgk_germany
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, type N95 (US)
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H Ji et al.
American journal of physiology. Regulatory, integrative and comparative physiology, 278(6), R1579-R1582 (2000-06-10)
Previous studies indicate that administration of the metabolic inhibitor, 2,5-anhydro-D-mannitol (2,5-AM) or methyl palmoxirate (MP), induces feeding behavior in rats by lowering hepatic energy status. Combined treatment with these agents synergistically increases food intake. The present study was designed to
C C Horn et al.
The American journal of physiology, 275(2 Pt 2), R448-R459 (1998-08-04)
Whether administration of 2,5-anhydro-D-mannitol (2,5-AM) or methyl palmoxirate (MP) elicits eating behavior in rats depends on the composition of the maintenance diet. To assess whether specific brain sites are involved in triggering the eating responses to these metabolic inhibitors, we
F Désy et al.
International journal of sports medicine, 20(1), 17-22 (1999-03-25)
The fructose analogue 2,5-anhydro-D-mannitol (2,5-AM) has been shown to act specifically in liver by decreasing liver ATP and by blocking glycogenolysis and gluconeogenesis. The present investigation was designed to determine the effects of the administration of 2,5-AM on pancreatic hormone
S Aiston et al.
Diabetologia, 43(5), 589-597 (2000-06-16)
The Zucker fatty fa/fa rat develops hyperinsulinaemia, insulin-resistance and severe obesity as a result of a homozygous mutation in the leptin receptor gene. The aim was to characterise the metabolic defect(s) in hepatocytes from fa/fa rats. Glucose metabolism and key
E Scharrer et al.
The American journal of physiology, 272(3 Pt 2), R874-R878 (1997-03-01)
Because 2,5-anhydro-D-mannitol (2,5-AM) seems to stimulate feeding by acting on the liver and because the hepatic membrane potential has been suggested to play an important role in control of feeding ("potentiostatic" hypothesis), we investigated the effect of 2,5-AM on the
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